Literature DB >> 22873530

Placebo-controlled trial of tofacitinib monotherapy in rheumatoid arthritis.

Roy Fleischmann1, Joel Kremer, John Cush, Hendrik Schulze-Koops, Carol A Connell, John D Bradley, David Gruben, Gene V Wallenstein, Samuel H Zwillich, Keith S Kanik.   

Abstract

BACKGROUND: Tofacitinib (CP-690,550) is a novel oral Janus kinase inhibitor that is being investigated as a targeted immunomodulator and disease-modifying therapy for rheumatoid arthritis.
METHODS: In this phase 3, double-blind, placebo-controlled, parallel-group, 6-month study, 611 patients were randomly assigned, in a 4:4:1:1 ratio, to 5 mg of tofacitinib twice daily, 10 mg of tofacitinib twice daily, placebo for 3 months followed by 5 mg of tofacitinib twice daily, or placebo for 3 months followed by 10 mg of tofacitinib twice daily. The primary end points, assessed at month 3, were the percentage of patients with at least a 20% improvement in the American College of Rheumatology scale (ACR 20), the change from baseline in Health Assessment Questionnaire-Disability Index (HAQ-DI) scores (which range from 0 to 3, with higher scores indicating greater disability), and the percentage of patients with a Disease Activity Score for 28-joint counts based on the erythrocyte sedimentation rate (DAS28-4[ESR]) of less than 2.6 (with scores ranging from 0 to 9.4 and higher scores indicating more disease activity).
RESULTS: At month 3, a higher percentage of patients in the tofacitinib groups than in the placebo groups met the criteria for an ACR 20 response (59.8% in the 5-mg tofacitinib group and 65.7% in the 10-mg tofacitinib group vs. 26.7% in the combined placebo groups, P<0.001 for both comparisons). The reductions from baseline in HAQ-DI scores were greater in the 5-mg and 10-mg tofacitinib groups than in the placebo groups (-0.50 and -0.57 points, respectively, vs. -0.19 points; P<0.001). The percentage of patients with a DAS28-4(ESR) of less than 2.6 was not significantly higher with tofacitinib than with placebo (5.6% and 8.7% in the 5-mg and 10-mg tofacitinib groups, respectively, and 4.4% with placebo; P=0.62 and P=0.10 for the two comparisons). Serious infections developed in six patients who were receiving tofacitinib. Common adverse events were headache and upper respiratory tract infection. Tofacitinib treatment was associated with elevations in low-density lipoprotein cholesterol levels and reductions in neutrophil counts.
CONCLUSIONS: In patients with active rheumatoid arthritis, tofacitinib monotherapy was associated with reductions in signs and symptoms of rheumatoid arthritis and improvement in physical function. (Funded by Pfizer; ORAL Solo ClinicalTrials.gov number, NCT00814307.).

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Year:  2012        PMID: 22873530     DOI: 10.1056/NEJMoa1109071

Source DB:  PubMed          Journal:  N Engl J Med        ISSN: 0028-4793            Impact factor:   91.245


  334 in total

1.  Tofacitinib as a novel salvage therapy for refractory T-cell large granular lymphocytic leukemia.

Authors:  B Bilori; S Thota; M J Clemente; B Patel; A Jerez; M Afable Ii; J P Maciejewski
Journal:  Leukemia       Date:  2015-10-09       Impact factor: 11.528

Review 2.  Tofacitinib.

Authors: 
Journal:  Aust Prescr       Date:  2015-10-14

3.  Small airway-on-a-chip enables analysis of human lung inflammation and drug responses in vitro.

Authors:  Kambez H Benam; Remi Villenave; Carolina Lucchesi; Antonio Varone; Cedric Hubeau; Hyun-Hee Lee; Stephen E Alves; Michael Salmon; Thomas C Ferrante; James C Weaver; Anthony Bahinski; Geraldine A Hamilton; Donald E Ingber
Journal:  Nat Methods       Date:  2015-12-21       Impact factor: 28.547

4.  Rash, Fever, and Pulmonary Hypertension in a 6-Year-Old Female.

Authors:  David Buchbinder; Gina A Montealegre Sanchez; Raphaela Goldbach-Mansky; Hermine Brunner; Andrew I Shulman
Journal:  Arthritis Care Res (Hoboken)       Date:  2018-04-02       Impact factor: 4.794

Review 5.  Novel Treatments in Lupus.

Authors:  Vasileios C Kyttaris
Journal:  Curr Rheumatol Rep       Date:  2017-03       Impact factor: 4.592

Review 6.  Janus kinases: an ideal target for the treatment of autoimmune diseases.

Authors:  Massimo Gadina
Journal:  J Investig Dermatol Symp Proc       Date:  2013-12

Review 7.  Interleukin-6: designing specific therapeutics for a complex cytokine.

Authors:  Christoph Garbers; Sylvia Heink; Thomas Korn; Stefan Rose-John
Journal:  Nat Rev Drug Discov       Date:  2018-05-04       Impact factor: 84.694

8.  Design and pharmacology of a highly specific dual FMS and KIT kinase inhibitor.

Authors:  Chao Zhang; Prabha N Ibrahim; Jiazhong Zhang; Elizabeth A Burton; Gaston Habets; Ying Zhang; Ben Powell; Brian L West; Bernice Matusow; Garson Tsang; Rafe Shellooe; Heidi Carias; Hoa Nguyen; Adhirai Marimuthu; Kam Y J Zhang; Angela Oh; Ryan Bremer; Clarence R Hurt; Dean R Artis; Guoxian Wu; Marika Nespi; Wayne Spevak; Paul Lin; Keith Nolop; Peter Hirth; Greg H Tesch; Gideon Bollag
Journal:  Proc Natl Acad Sci U S A       Date:  2013-03-14       Impact factor: 11.205

Review 9.  The current state of the art for biological therapies and new small molecules in inflammatory bowel disease.

Authors:  Sudarshan Paramsothy; Adam K Rosenstein; Saurabh Mehandru; Jean-Frederic Colombel
Journal:  Mucosal Immunol       Date:  2018-06-15       Impact factor: 7.313

Review 10.  Janus kinase inhibitors in autoimmune diseases.

Authors:  John J O'Shea; Apostolos Kontzias; Kunihiro Yamaoka; Yoshiya Tanaka; Arian Laurence
Journal:  Ann Rheum Dis       Date:  2013-04       Impact factor: 19.103

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