Literature DB >> 22808506

Neutrophil apoptosis induction by tick-borne encephalitis virus.

N G Plekhova1, L M Somova, I N Lyapun, N V Krylova, G N Leonova.   

Abstract

Tick-borne encephalitis virus infects neutrophils and induces their apoptosis, judging from moderate increase of succinate dehydrogenase activity and a trend to anaerobic energy production in neutrophils infected with the virus (shown by an increase of lactate dehydrogenase activity).

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Year:  2012        PMID： 22808506     DOI： 10.1007/s10517-012-1655-2

Source DB:  PubMed          Journal:  Bull Exp Biol Med        ISSN： 0007-4888            Impact factor:   0.804


  3 in total

1.  Dual Function of Ccr5 during Langat Virus Encephalitis: Reduction in Neutrophil-Mediated Central Nervous System Inflammation and Increase in T Cell-Mediated Viral Clearance.

Authors:  Daniela Michlmayr; Susana V Bardina; Carlos A Rodriguez; Alexander G Pletnev; Jean K Lim
Journal:  J Immunol       Date:  2016-04-29       Impact factor: 5.422

2.  Upregulated Intrathecal Expression of VEGF-A and Long Lasting Global Upregulation of Proinflammatory Immune Mediators in Vaccine Breakthrough Tick-Borne Encephalitis.

Authors:  Miša Pavletič; Misa Korva; Nataša Knap; Petra Bogovič; Lara Lusa; Klemen Strle; Mirijam Nahtigal Klevišar; Tomaž Vovko; Janez Tomažič; Stanka Lotrič-Furlan; Franc Strle; Tatjana Avšič-Županc
Journal:  Front Cell Infect Microbiol       Date:  2021-07-01       Impact factor: 5.293

3.  The three subtypes of tick-borne encephalitis virus induce encephalitis in a natural host, the bank vole (Myodes glareolus).

Authors:  Elina Tonteri; Anja Kipar; Liina Voutilainen; Sirkka Vene; Antti Vaheri; Olli Vapalahti; Åke Lundkvist
Journal:  PLoS One       Date:  2013-12-13       Impact factor: 3.240
  3 in total

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