Literature DB >> 22777068

[New kinase inhibitors].

A Rubbert-Roth1.   

Abstract

Treatment of rheumatoid arthritis (RA) has dramatically changed during the last 15 years. A limited number of conventional disease-modifying antirheumatic drugs (DMARD) in combination with non-steroid anti-inflammatory drugs (NSAID) and corticosteroids are facing a variety of biologics that are increasingly being used. Because of the high costs of biologics as well as the necessity for subcutaneous or intravenous administration, there is currently a growing interest in new and potent oral compounds such as the small molecules. Inflammatory pathways and mechanisms in signal transduction have been characterized in detail. Instead of neutralizing the action of a proinflammatory cytokine by antagonizing its biologic effect by an antibody, these small molecules interfere with the intracellular pathways of the inflammatory cascade. Intracellular kinases are among these enzymes which are crucially involved in intracellular signal transduction. Kinase inhibitors have been successfully used within the last few years in the treatment of various hematological malignancies, such as imatinib in patients with chronic myeloid leukemia. More recently, the Janus kinase (JAK) inhibitor tofacitinib has been evaluated as a potential new treatment option in RA and is awaiting approval. While an overview about JAK inhibition will be given elsewhere, other inhibitors such as spleen tyrosine kinase (Syk) inhibitor, mitogen-activated protein kinase (MAPK) inhibitor and Bruton's tyrosine kinase (Btk) inhibitor are currently in preclinical and clinical development.

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Year:  2012        PMID: 22777068     DOI: 10.1007/s00393-011-0880-9

Source DB:  PubMed          Journal:  Z Rheumatol        ISSN: 0340-1855            Impact factor:   1.372


  28 in total

1.  Differential tissue expression and activation of p38 MAPK alpha, beta, gamma, and delta isoforms in rheumatoid arthritis.

Authors:  Adelheid Korb; Makiyeh Tohidast-Akrad; Erdal Cetin; Roland Axmann; Josef Smolen; Georg Schett
Journal:  Arthritis Rheum       Date:  2006-09

2.  c-Jun N-terminal kinase is required for metalloproteinase expression and joint destruction in inflammatory arthritis.

Authors:  Z Han; D L Boyle; L Chang; B Bennett; M Karin; L Yang; A M Manning; G S Firestein
Journal:  J Clin Invest       Date:  2001-07       Impact factor: 14.808

3.  Activation of p38 MAPK is a key step in tumor necrosis factor-mediated inflammatory bone destruction.

Authors:  Jochen Zwerina; Silvia Hayer; Kurt Redlich; Klaus Bobacz; Giorgos Kollias; Josef S Smolen; Georg Schett
Journal:  Arthritis Rheum       Date:  2006-02

4.  Inhibition of fms-like tyrosine kinase 3 alleviates experimental arthritis by reducing formation of dendritic cells and antigen presentation.

Authors:  Mats Dehlin; Sofia Andersson; Malin Erlandsson; Mikael Brisslert; Maria Bokarewa
Journal:  J Leukoc Biol       Date:  2011-07-19       Impact factor: 4.962

5.  Evaluation of the efficacy and safety of pamapimod, a p38 MAP kinase inhibitor, in a double-blind, methotrexate-controlled study of patients with active rheumatoid arthritis.

Authors:  Stanley B Cohen; Tien-Tsai Cheng; Vishala Chindalore; Nemanja Damjanov; Ruben Burgos-Vargas; Patricia Delora; Kathleen Zimany; Helen Travers; John P Caulfield
Journal:  Arthritis Rheum       Date:  2009-02

6.  Induction of arthritis by single monoclonal IgG anti-collagen type II antibodies and enhancement of arthritis in mice lacking inhibitory FcgammaRIIB.

Authors:  Kutty Selva Nandakumar; Maria Andrén; Pernilla Martinsson; Estelle Bajtner; Silvia Hellström; Rikard Holmdahl; Sandra Kleinau
Journal:  Eur J Immunol       Date:  2003-08       Impact factor: 5.532

7.  Imatinib mesylate both prevents and treats the arthritis induced by type II collagen antibody in mice.

Authors:  Kensuke Koyama; Kyosuke Hatsushika; Takashi Ando; Michitomo Sakuma; Masanori Wako; Ryohei Kato; Hirotaka Haro; Hajime Sugiyama; Yoshiki Hamada; Hideoki Ogawa; Atsuhito Nakao
Journal:  Mod Rheumatol       Date:  2007-08-20       Impact factor: 3.023

8.  Prevention of the onset and progression of collagen-induced arthritis in rats by the potent p38 mitogen-activated protein kinase inhibitor FR167653.

Authors:  Masataka Nishikawa; Akira Myoui; Tetsuya Tomita; Koichiro Takahi; Akihide Nampei; Hideki Yoshikawa
Journal:  Arthritis Rheum       Date:  2003-09

9.  Selective tyrosine kinase inhibition by imatinib mesylate for the treatment of autoimmune arthritis.

Authors:  Ricardo T Paniagua; Orr Sharpe; Peggy P Ho; Steven M Chan; Anna Chang; John P Higgins; Beren H Tomooka; Fiona M Thomas; Jason J Song; Stuart B Goodman; David M Lee; Mark C Genovese; Paul J Utz; Lawrence Steinman; William H Robinson
Journal:  J Clin Invest       Date:  2006-09-14       Impact factor: 14.808

10.  The Btk tyrosine kinase is a major target of the Bcr-Abl inhibitor dasatinib.

Authors:  Oliver Hantschel; Uwe Rix; Uwe Schmidt; Tilmann Bürckstümmer; Michael Kneidinger; Gregor Schütze; Jacques Colinge; Keiryn L Bennett; Wilfried Ellmeier; Peter Valent; Giulio Superti-Furga
Journal:  Proc Natl Acad Sci U S A       Date:  2007-08-07       Impact factor: 11.205

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