Literature DB >> 22696515

Availability of energetic substrates and exercise performance in heart failure with or without diabetes.

Vojtech Melenovsky1, Martin Kotrc, Jan Polak, Terezie Pelikanova, Bela Bendlova, Monika Cahova, Ivan Malek, Petr Jarolim, Ludmila Kazdova, Josef Kautzner.   

Abstract

AIMS: The goal of the study was to examine whether resting or post-exercise metabolic substrate levels are associated with differential exercise performance and long-term outcome in control subjects or heart failure (HF) patients with or without type 2 diabetes mellitus (DM). METHODS AND
RESULTS: Twenty five healthy controls matched with 97 patients with stable advanced HF were prospectively enrolled. Exercise capacity, age, gender, and HF aetiology were balanced between HFDM- and HFDM+ groups. Subjects underwent maximal bicycle spiroergometry with blood sampling to measure metabolites and neurohormones before and immediately after the exercise. HFDM+ patients had increased free fatty acids, glucose, and β-hydroxybutyrate compared with controls. HFDM+ patients had higher baseline copeptin (24 ± 16 vs. 17 ± 13 pmol/L, P < 0.05) but otherwise showed similar neurohumoral activation and exercise response to HFDM- patients. Peak oxygen consumption (VO(2)) was unrelated to post-exercise free fatty acids, glucose, lactate, or glycerol, but strongly correlated with post-exercise pyruvate (in all: r = 0.62, P < 0.001). During the next 17 ± 10 months, 36% of HF patients experienced an adverse event (death, urgent transplantation, or assist device insertion). From metabolic factors, only post-exercise glucose [hazard ratio (HR) 1.28, P = 0.04), total body fat (HR 0.58, P < 0.001), and the presence of DM (HR 1.98, P = 0.04) were predictive of the outcome.
CONCLUSIONS: With the exception of pyruvate, acute changes of metabolic substrates are not related to cardiac performance in HF, regardless of diabetic status. Inhibition of body fat depletion, attenuation of stress-related hyperglycaemia, or increasing dynamics of plasma pyruvate may represent therapeutic targets in advanced HF.

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Year:  2012        PMID: 22696515     DOI: 10.1093/eurjhf/hfs080

Source DB:  PubMed          Journal:  Eur J Heart Fail        ISSN: 1388-9842            Impact factor:   15.534


  6 in total

Review 1.  Pyruvate and Metabolic Flexibility: Illuminating a Path Toward Selective Cancer Therapies.

Authors:  Kristofor A Olson; John C Schell; Jared Rutter
Journal:  Trends Biochem Sci       Date:  2016-02-10       Impact factor: 13.807

2.  Adrenergic activation, fuel substrate availability, and insulin resistance in patients with congestive heart failure.

Authors:  Nir Uriel; Jose Gonzalez-Costello; Andrea Mignatti; Kerry A Morrison; Nadav Nahumi; Paolo C Colombo; Ulrich P Jorde
Journal:  JACC Heart Fail       Date:  2013-08-05       Impact factor: 12.035

3.  Metabolomic fingerprint of heart failure with preserved ejection fraction.

Authors:  Beshay N Zordoky; Miranda M Sung; Justin Ezekowitz; Rupasri Mandal; Beomsoo Han; Trent C Bjorndahl; Souhaila Bouatra; Todd Anderson; Gavin Y Oudit; David S Wishart; Jason R B Dyck
Journal:  PLoS One       Date:  2015-05-26       Impact factor: 3.240

4.  Glucose Homeostasis, Pancreatic Endocrine Function, and Outcomes in Advanced Heart Failure.

Authors:  Vojtech Melenovsky; Jan Benes; Janka Franekova; Jan Kovar; Barry A Borlaug; Marketa Segetova; Andrea Tura; Tereza Pelikanova
Journal:  J Am Heart Assoc       Date:  2017-08-07       Impact factor: 5.501

5.  Predictive value of plasma copeptin level for the risk and mortality of heart failure: a meta-analysis.

Authors:  Jian-Jun Yan; Ying Lu; Zheng-Ping Kuai; Yong-Hong Yong
Journal:  J Cell Mol Med       Date:  2017-02-28       Impact factor: 5.310

Review 6.  Loss of Metabolic Flexibility in the Failing Heart.

Authors:  Qutuba G Karwi; Golam M Uddin; Kim L Ho; Gary D Lopaschuk
Journal:  Front Cardiovasc Med       Date:  2018-06-06
  6 in total

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