Literature DB >> 22619063

Refinement of stopping rules during treatment of hepatitis C genotype 1 infection with boceprevir and peginterferon/ribavirin.

Ira M Jacobson1, Patrick Marcellin, Stefan Zeuzem, Mark S Sulkowski, Rafael Esteban, Fred Poordad, Savino Bruno, Margaret H Burroughs, Lisa D Pedicone, Navdeep Boparai, Weiping Deng, Mark J DiNubile, Keith M Gottesdiener, Clifford A Brass, Janice K Albrecht, Jean-Pierre Bronowicki.   

Abstract

UNLABELLED: In comparison with peginterferon/ribavirin alone, boceprevir with peginterferon/ribavirin significantly improves sustained virological response (SVR) rates in patients with chronic hepatitis C virus (HCV) genotype 1 infections, but treatment failure remains a significant problem. Using phase 3 trial databases, we sought to develop stopping rules for patients destined to fail boceprevir-based combination therapy in order to minimize drug toxicity, resistance, and costs in the face of ultimate futility. Exploratory post hoc analyses using data from the Serine Protease Inhibitor Therapy 2 (SPRINT-2) study (treatment-naive patients) and the Retreatment With HCV Serine Protease Inhibitor Boceprevir and Pegintron/Rebetol 2 (RESPOND-2) study (treatment-experienced patients) were undertaken to determine whether protocol-specified stopping rules (detectable HCV RNA at week 24 for SPRINT-2 and at week 12 for RESPOND-2) could be refined and harmonized. In SPRINT-2, a week 12 rule with an HCV RNA cutoff of ≥ 100 IU/mL would have discontinued therapy in 65 of 195 failures (sensitivity = 33%) without sacrificing a single SVR among 475 successes (specificity = 100%). Viral variants emerged after week 12 in 36 of the 49 evaluable patients (73%) who would have discontinued at week 12 using a ≥ 100 IU/mL stopping rule. In RESPOND-2, five of six patients with week 12 HCV RNA levels between the lower limit of detection (9.3 IU/mL) and the lower limit of quantification (25 IU/mL) who continued therapy despite the protocol-stipulated futility rule achieved SVR; one additional patient with a week 12 HCV RNA level of 148 IU/mL also continued therapy, had undetectable HCV RNA at week 16, and attained SVR.
CONCLUSION: Although a stopping rule of detectable HCV RNA at week 12 would have forfeited some SVR cases, week 12 HCV RNA levels ≥ 100 IU/mL almost universally predicted a failure to achieve SVR in both treatment-naive and treatment-experienced patients. In boceprevir recipients, the combination of 2 stopping rules-an HCV RNA level ≥ 100 IU/mL at week 12 and detectable HCV RNA at week 24--maximized the early discontinuation of futile therapy and minimized premature treatment discontinuation.
Copyright © 2012 American Association for the Study of Liver Diseases.

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Year:  2012        PMID: 22619063     DOI: 10.1002/hep.25865

Source DB:  PubMed          Journal:  Hepatology        ISSN: 0270-9139            Impact factor:   17.425


  16 in total

1.  Virological outcomes and treatment algorithms utilisation in observational study of patients with chronic hepatitis C treated with boceprevir or telaprevir.

Authors:  R K Sterling; A Kuo; V K Rustgi; M S Sulkowski; T G Stewart; J M Fenkel; H El-Genaidi; M A Mah'moud; G M Abraham; P W Stewart; L Akushevich; D R Nelson; M W Fried; A M Di Bisceglie
Journal:  Aliment Pharmacol Ther       Date:  2015-01-28       Impact factor: 8.171

Review 2.  A new era in the treatment of chronic hepatitis C infection.

Authors:  Dinesh Jothimani; George M Chandy; Hari Conjeevaram
Journal:  Indian J Gastroenterol       Date:  2012-09-28

Review 3.  Hepatitis C.

Authors:  Daniel P Webster; Paul Klenerman; Geoffrey M Dusheiko
Journal:  Lancet       Date:  2015-02-14       Impact factor: 79.321

4.  The Cobas AmpliPrep/Cobas TaqMan HCV test, version 2.0, real-time PCR assay accurately quantifies hepatitis C virus genotype 4 RNA.

Authors:  Stéphane Chevaliez; Magali Bouvier-Alias; Christophe Rodriguez; Alexandre Soulier; Jean-Dominique Poveda; Jean-Michel Pawlotsky
Journal:  J Clin Microbiol       Date:  2013-01-16       Impact factor: 5.948

Review 5.  Individualization of chronic hepatitis C treatment according to the host characteristics.

Authors:  Nikolaos K Gatselis; Kalliopi Zachou; Asterios Saitis; Maria Samara; George N Dalekos
Journal:  World J Gastroenterol       Date:  2014-03-21       Impact factor: 5.742

6.  Importance of virological response in the early stage of telaprevir-based triple therapy for hepatitis C.

Authors:  Satoshi Hiramine; Norihiro Furusyo; Eiichi Ogawa; Makoto Nakamuta; Eiji Kajiwara; Hideyuki Nomura; Kazufumi Dohmen; Kazuhiro Takahashi; Takeaki Satoh; Koichi Azuma; Akira Kawano; Toshimasa Koyanagi; Kazuhiro Kotoh; Shinji Shimoda; Jun Hayashi
Journal:  World J Hepatol       Date:  2015-11-18

7.  The Usefulness of Defining Rapid Virological Response by a Very Sensitive Assay (TMA) during Treatment of HCV Genotype 2/3 Infection.

Authors:  Olav Dalgard; Michelle Martinot-Peignoux; Hans Verbaan; Kristian Bjøro; Helmer Ring-Larsen; Patrick Marcellin
Journal:  PLoS One       Date:  2015-08-28       Impact factor: 3.240

Review 8.  Therapeutic algorithms for chronic hepatitis C in the DAA era during the current economic crisis: whom to treat? How to treat? When to treat?

Authors:  Salvatore Petta; Antonio Craxì
Journal:  BMC Infect Dis       Date:  2012-11-12       Impact factor: 3.090

9.  Boceprevir and personalized medicine in hepatitis C virus infection.

Authors:  François Habersetzer; Céline Leboeuf; Michel Doffoël; Thomas F Baumert
Journal:  Pharmgenomics Pers Med       Date:  2012-09-26

10.  Rapid Prediction of Treatment Futility of Boceprevir with Peginterferon-Ribavirin for Taiwanese Treatment Experienced Hepatitis C Virus Genotype 1-Infected Patients.

Authors:  Chi-Chieh Yang; Wei-Lun Tsai; Wei-Wen Su; Chung-Feng Huang; Pin-Nan Cheng; Ching-Chu Lo; Kuo-Chih Tseng; Lein-Ray Mo; Chun-Hsiang Wang; Shih-Jer Hsu; Hsueh-Chou Lai; Chien-Wei Su; Chun-Jen Liu; Cheng-Yuan Peng; Ming-Lung Yu
Journal:  PLoS One       Date:  2015-09-14       Impact factor: 3.240

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