Literature DB >> 22614058

Lipopolysaccharide-induced chorioamnionitis is confined to one amniotic compartment in twin pregnant sheep.

Markus Gantert1, Reint K Jellema, Heike Heineman, Julia Gantert, Jennifer J P Collins, Matthias Seehase, Verena A C Lambermont, Alexander Keck, Yves Garnier, Luc J I Zimmermann, Mahmed Kadyrov, A W Danilo Gavilanes, Boris W Kramer.   

Abstract

BACKGROUND: Chorioamnionitis is a major risk factor for preterm birth in multifetal pregnancies. However, there is little clinical data whether chorioamnionitis is restricted to one amniotic compartment in multifetal pregnancies.
OBJECTIVE: To explore whether chorioamnionitis is confined to the exposed compartment and does not cross to the unaffected fetus in twin pregnancy.
METHODS: In twin pregnant sheep, one of the twins was exposed to either 2 or 14 days of intra-amniotic lipopolysaccharide (LPS) while the co-twin was exposed to either 2 or 14 days of intra-amniotic saline (n = 3 for each exposure). Singletons were included in this study to compare the grade of inflammation with twins. All fetuses were delivered at 125 days of gestation (term = 150 days). Chorioamnionitis was confirmed by histological examination. Lung inflammation was assessed by cell count in bronchoalveolar lavage. Lung compliance was assessed at 40 cm H(2)O. Results were compared using analysis of variance (ANOVA) with a post-hoc Tukey analysis.
RESULTS: Inflammation in placenta, membranes and lung of LPS-exposed twins was significantly higher after 2 and 14 days of exposure when compared to the saline-exposed co-twins. Lung compliance in LPS-exposed twins was significantly increased after 14 days when compared to saline-exposed co-twins. Intrauterine LPS exposure increased lung compliance and inflammation in the membranes, placenta and lung to the same extent in twins as in singletons.
CONCLUSION: In twin pregnant sheep, inflammation of the membranes, placenta and fetal lung was strictly limited to the exposed fetus in the amniotic compartment in which the LPS was injected.
Copyright © 2012 S. Karger AG, Basel.

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Year:  2012        PMID: 22614058     DOI: 10.1159/000338015

Source DB:  PubMed          Journal:  Neonatology        ISSN: 1661-7800            Impact factor:   4.035


  5 in total

1.  Pulmonary Morbidity in Infancy after Exposure to Chorioamnionitis in Late Preterm Infants.

Authors:  Karen M McDowell; Alan H Jobe; Matthew Fenchel; William D Hardie; Tate Gisslen; Lisa R Young; Claire A Chougnet; Stephanie D Davis; Suhas G Kallapur
Journal:  Ann Am Thorac Soc       Date:  2016-06

2.  Characterization of the fetal blood transcriptome and proteome in maternal anti-fetal rejection: evidence of a distinct and novel type of human fetal systemic inflammatory response.

Authors:  Joonho Lee; Roberto Romero; Tinnakorn Chaiworapongsa; Zhong Dong; Adi L Tarca; Yi Xu; Po Jen Chiang; Juan Pedro Kusanovic; Sonia S Hassan; Lami Yeo; Bo Hyun Yoon; Nandor Gabor Than; Chong Jai Kim
Journal:  Am J Reprod Immunol       Date:  2013-07-30       Impact factor: 3.886

3.  Intrauterine inflammation alters fetal cardiopulmonary and cerebral haemodynamics in sheep.

Authors:  Robert Galinsky; Stuart B Hooper; Graeme R Polglase; Timothy J M Moss
Journal:  J Physiol       Date:  2013-07-22       Impact factor: 5.182

4.  Lipopolysaccharide and soluble CD14 in cord blood plasma are associated with prematurity and chorioamnionitis.

Authors:  Denise G Martinez-Lopez; Nicholas T Funderburg; Adam Cerissi; Reema Rifaie; Laura Aviles-Medina; Braulio J Llorens-Bonilla; John Sleasman; Angel A Luciano
Journal:  Pediatr Res       Date:  2013-10-17       Impact factor: 3.756

5.  Intra-Amniotic LPS Induced Region-Specific Changes in Presynaptic Bouton Densities in the Ovine Fetal Brain.

Authors:  Eveline Strackx; Reint K Jellema; Rebecca Rieke; Ruth Gussenhoven; Johan S H Vles; Boris W Kramer; Antonio W D Gavilanes
Journal:  Biomed Res Int       Date:  2015-08-31       Impact factor: 3.411

  5 in total

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