Literature DB >> 22591424

Multiple VEGF family members are simultaneously expressed in ovarian cancer: a proposed model for bevacizumab resistance.

Arne R M van der Bilt1, Ate G J van der Zee, Elisabeth G E de Vries, Steven de Jong, Hetty Timmer-Bosscha, Klaske A ten Hoor, Wilfred F A den Dunnen, Harry Hollema, Anna K L Reyners.   

Abstract

OBJECTIVE: Insight into the expression of multiple vascular endothelial growth factor (VEGF) family members can support the implementation of anti-angiogenic therapy. This study aimed to assess VEGF family member expression in ovarian cancers and related omental metastases.
METHODS: Tissue microarrays encompassing 270 primary cancers and 112 paired metastases were immunostained for VEGF-A, VEGF-B, VEGF-C and VEGF-D. Staining intensities were categorized as absent, weak, moderate or strong. Expression was related to clinicopathological characteristics and survival.
RESULTS: Immunohistochemical positivity (defined as moderate or strong expression) was observed for VEGF-A in 90%, VEGF-B in 4%, VEGF-C in 41% and VEGF-D in 55% of the primary ovarian cancers. VEGF-A expression correlated with VEGF-C and VEGF-D expression (P < 0.01). Simultaneous positivity for VEGF-A and VEGF-C or VEGF-D was observed in 38% and 54% of the cancers, respectively. Metastases showed positivity for VEGF-A in 78%, VEGF-B in 5%, VEGF-C in 26% and VEGF-D in 45% of cases. VEGF family member expression showed no independent prognostic significance in multivariate survival analysis.
CONCLUSION: VEGF-A, VEGF-C and VEGF-D are widely and often simultaneously expressed in ovarian cancer, which may contribute to bevacizumab resistance. Measuring their expression could support a rational, individualized choice of anti-angiogenic therapy and might be of predictive value. Studies are warranted to determine whether combinatorial analysis of VEGF family member expression can be used to predict anti-angiogenic drug efficacy.

Entities:  

Mesh:

Substances:

Year:  2012        PMID: 22591424     DOI: 10.2174/138161212802002661

Source DB:  PubMed          Journal:  Curr Pharm Des        ISSN: 1381-6128            Impact factor:   3.116


  10 in total

1.  Hypoxia-regulated overexpression of soluble VEGFR2 controls angiogenesis and inhibits tumor growth.

Authors:  Guillaume Collet; Nathalie Lamerant-Fayel; Magdalena Tertil; Bouchra El Hafny-Rahbi; Jacek Stepniewski; Alan Guichard; Alexandra Foucault-Collet; Krzysztof Klimkiewicz; Stéphane Petoud; Agata Matejuk; Catherine Grillon; Alicja Jozkowicz; Jozef Dulak; Claudine Kieda
Journal:  Mol Cancer Ther       Date:  2013-10-29       Impact factor: 6.261

2.  VEGFR3 inhibition chemosensitizes ovarian cancer stemlike cells through down-regulation of BRCA1 and BRCA2.

Authors:  Jaeyoung J Lim; Kun Yang; Barbie Taylor-Harding; W Ruprecht Wiedemeyer; Ronald J Buckanovich
Journal:  Neoplasia       Date:  2014-04       Impact factor: 5.715

3.  Epithelial ovarian cancer-induced angiogenic phenotype of human omental microvascular endothelial cells may occur independently of VEGF signaling.

Authors:  Boleslaw K Winiarski; Katarzyna I Wolanska; Srijana Rai; Tahanver Ahmed; Nigel Acheson; Nicholas J Gutowski; Jacqueline L Whatmore
Journal:  Transl Oncol       Date:  2013-12-01       Impact factor: 4.243

4.  PG545 enhances anti-cancer activity of chemotherapy in ovarian models and increases surrogate biomarkers such as VEGF in preclinical and clinical plasma samples.

Authors:  Boris Winterhoff; Luisa Freyer; Edward Hammond; Shailendra Giri; Susmita Mondal; Debarshi Roy; Attila Teoman; Sally A Mullany; Robert Hoffmann; Antonia von Bismarck; Jeremy Chien; Matthew S Block; Michael Millward; Darryn Bampton; Keith Dredge; Viji Shridhar
Journal:  Eur J Cancer       Date:  2015-03-05       Impact factor: 9.162

5.  Dual HER/VEGF receptor targeting inhibits in vivo ovarian cancer tumor growth.

Authors:  Marc A Becker; Thahir Farzan; Sean C Harrington; James W Krempski; S John Weroha; Xiaonan Hou; Kimberly R Kalli; Tai W Wong; Paul Haluska
Journal:  Mol Cancer Ther       Date:  2013-10-15       Impact factor: 6.261

6.  Predictive Blood-Based Biomarkers in Patients with Epithelial Ovarian Cancer Treated with Carboplatin and Paclitaxel with or without Bevacizumab: Results from GOG-0218.

Authors:  Angeles Alvarez Secord; Kirsten Bell Burdett; Kouros Owzar; David Tritchler; Alexander B Sibley; Yingmiao Liu; Mark D Starr; J Chris Brady; Heather A Lankes; Herbert I Hurwitz; Robert S Mannel; Krishnansu S Tewari; David M O'Malley; Heidi Gray; Jamie N Bakkum-Gamez; Keiichi Fujiwara; Matthew Boente; Wei Deng; Robert A Burger; Michael J Birrer; Andrew B Nixon
Journal:  Clin Cancer Res       Date:  2020-01-09       Impact factor: 12.531

7.  Morpholino-Mediated Isoform Modulation of Vascular Endothelial Growth Factor Receptor-2 (VEGFR2) Reduces Colon Cancer Xenograft Growth.

Authors:  Brian C Stagg; Hironori Uehara; Nathan Lambert; Ruju Rai; Isha Gupta; Bryce Radmall; Taylor Bates; Balamurali K Ambati
Journal:  Cancers (Basel)       Date:  2014-11-26       Impact factor: 6.639

Review 8.  Angiogenesis: A Pivotal Therapeutic Target in the Drug Development of Gynecologic Cancers.

Authors:  Lawrence Kasherman; Shiru Lucy Liu; Katherine Karakasis; Stephanie Lheureux
Journal:  Cancers (Basel)       Date:  2022-02-22       Impact factor: 6.639

Review 9.  The Potential Role of the Proteases Cathepsin D and Cathepsin L in the Progression and Metastasis of Epithelial Ovarian Cancer.

Authors:  Md Zahidul Islam Pranjol; Nicholas Gutowski; Michael Hannemann; Jacqueline Whatmore
Journal:  Biomolecules       Date:  2015-11-20

10.  Fe-MIL-101 exhibits selective cytotoxicity and inhibition of angiogenesis in ovarian cancer cells via downregulation of MMP.

Authors:  Jiaqiang Wang; Daomei Chen; Bin Li; Jiao He; Deliang Duan; Dandan Shao; Minfang Nie
Journal:  Sci Rep       Date:  2016-05-18       Impact factor: 4.379

  10 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.