Literature DB >> 22516899

Copy number changes on the X chromosome in women with and without highly skewed X-chromosome inactivation.

V Jobanputra1, B Levy, A Kinney, S Brown, M Shirazi, C Yu, J Kline, D Warburton.   

Abstract

AIM: To test the hypothesis that microdeletions or microduplications below the resolution of a standard karyotype may be a significant cause of highly skewed X-inactivation (HSXI) in women without a cytogenetically detected X-chromosome anomaly.
METHODS: Cases were women with HSXI, defined as ≥85% of cells in a blood sample with the same active allele at the HUMARA locus. The skewing in controls ranged from 50 to <75%. We performed an SNP microarray analysis using the Affymetrix 6.0 platform for 45 cases and 45 controls.
RESULTS: Cases and controls did not differ in the frequency of X-chromosome copy number changes ≥100 kb or in the frequency of copy number changes that contained genes. However, one woman with HSXI >90% in blood and left and right buccal smears had a 5.5-Mb deletion in Xp22.2p22.1. This deletion could affect the viability of male conceptions and may have led to the dysmorphology found in female carriers.
CONCLUSION: HSXI in a blood sample is rarely due to X-chromosome copy number changes detectable by microarray.
Copyright © 2012 S. Karger AG, Basel.

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Year:  2012        PMID: 22516899      PMCID: PMC4315938          DOI: 10.1159/000337920

Source DB:  PubMed          Journal:  Cytogenet Genome Res        ISSN: 1424-8581            Impact factor:   1.636


  21 in total

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2.  Skewed X chromosome inactivation and trisomic spontaneous abortion: no association.

Authors:  Dorothy Warburton; Jennie Kline; Ann Kinney; Chih-Yu Yu; Bruce Levin; Stephen Brown
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3.  A promoter mutation in the XIST gene in two unrelated families with skewed X-chromosome inactivation.

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4.  Uneven X inactivation in a female monozygotic twin pair with Fabry disease and discordant expression of a novel mutation in the alpha-galactosidase A gene.

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Journal:  J Med Genet       Date:  1996-08       Impact factor: 6.318

5.  Familial skewed X inactivation: a molecular trait associated with high spontaneous-abortion rate maps to Xq28.

Authors:  E Pegoraro; J Whitaker; P Mowery-Rushton; U Surti; M Lanasa; E P Hoffman
Journal:  Am J Hum Genet       Date:  1997-07       Impact factor: 11.025

6.  Primordial cell pool size and lineage relationships of five human cell types.

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7.  X chromosome inactivation in fibroblasts of mentally retarded female carriers of the fragile site Xq27.3: application of the probe M27 beta to evaluate X inactivation status.

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8.  Preferential X-chromosome inactivation in women with idiopathic recurrent pregnancy loss.

Authors:  S Uehara; M Hashiyada; K Sato; Y Sato; K Fujimori; K Okamura
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9.  Tissue specificity of X-chromosome inactivation patterns.

Authors:  R E Gale; H Wheadon; P Boulos; D C Linch
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10.  Skewed X-chromosome inactivation is associated with trisomy in women ascertained on the basis of recurrent spontaneous abortion or chromosomally abnormal pregnancies.

Authors:  C L Beever; M D Stephenson; M S Peñaherrera; R H Jiang; D K Kalousek; M Hayden; L Field; C J Brown; W P Robinson
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  2 in total

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Journal:  Mol Syndromol       Date:  2017-09-07

2.  Demonstration of a novel Xp22.2 microdeletion as the cause of familial extreme skewing of X-inactivation utilizing case-parent trio SNP microarray analysis.

Authors:  Jane A Mason; Hnin T Aung; Adayapalam Nandini; Rickie G Woods; David J Fairbairn; John A Rowell; David Young; Rachel D Susman; Simon A Brown; Valentine J Hyland; Jeremy D Robertson
Journal:  Mol Genet Genomic Med       Date:  2018-02-28       Impact factor: 2.183

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