Literature DB >> 22425623

Preventing and reversing the cellular consequences of Z alpha-1 antitrypsin accumulation by targeting s4A.

Sam Alam1, Jicun Wang, Sabina Janciauskiene, Ravi Mahadeva.   

Abstract

BACKGROUND & AIMS: The Z variant (Glu342Lys) of α(1)-antitrypsin (AT) polymerizes and accumulates in the hepatocyte endoplasmic reticulum (ER) predisposing to neonatal hepatitis and liver cirrhosis. The resultant secretory defect leaves the lungs vulnerable to elastolysis and early-onset emphysema. Our aim in this study was to evaluate the effect of targeting strand 4a (s4A) as a strategy to inhibit polymerization and restore plasma secretion.
METHODS: HEK293 cells and HepG2 cells were transfected with Z-AT (Z-AT cells) or control M-AT (M-AT cells). The effect of Ac-TTAI-NH(2) (4M), Ac-FLEAIG-NH(2) (6M), and Ac-SEAAASTAVVIA-NH(2) (12M) on preventing and reversing intracellular Z-AT polymers and secretion of AT was evaluated by pulse-chase/immunoprecipitation, ELISA, and immunoblot with a polymer-specific antibody (ATZII). The ER overload response was assessed by RT-PCR for PERK, calnexin, and RGS16, and ELISA for NF-κB, IL-6, and IL-8.
RESULTS: All peptides prevented the intracellular accumulation of Z-AT (4M>6M>12M) in comparison with control peptides, with detection of the AT-Inhibitor complex in inclusion bodies. In so doing, 4M also significantly increased the concentration of secreted Z-AT and the elastase inhibitory activity. Furthermore, the 4M peptide was able to reverse the intracellular aggregation of Z-AT. The ER accumulation of Z-AT was shown to induce PERK-dependent NF-κB, IL-6, IL-8, and RGS16 and calnexin; all of which could be abrogated effectively by 4M. 4M had no effect on apoptosis or cell viability.
CONCLUSIONS: These findings are the first evidence that targeting s4A can prevent the cellular accumulation and deleterious effects of Z-AT and restore its plasma concentrations. As such, this is a major step towards treatment of patients with Z-AT-related disease.
Copyright © 2012 European Association for the Study of the Liver. Published by Elsevier B.V. All rights reserved.

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Year:  2012        PMID: 22425623     DOI: 10.1016/j.jhep.2012.02.025

Source DB:  PubMed          Journal:  J Hepatol        ISSN: 0168-8278            Impact factor:   25.083


  9 in total

1.  Antisense oligonucleotide treatment ameliorates alpha-1 antitrypsin-related liver disease in mice.

Authors:  Shuling Guo; Sheri L Booten; Mariam Aghajan; Gene Hung; Chenguang Zhao; Keith Blomenkamp; Danielle Gattis; Andrew Watt; Susan M Freier; Jeffery H Teckman; Michael L McCaleb; Brett P Monia
Journal:  J Clin Invest       Date:  2013-12-20       Impact factor: 14.808

2.  Histone deacetylase inhibitor (HDACi) suberoylanilide hydroxamic acid (SAHA)-mediated correction of α1-antitrypsin deficiency.

Authors:  Marion Bouchecareilh; Darren M Hutt; Patricia Szajner; Terence R Flotte; William E Balch
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3.  Z α1-antitrypsin confers a proinflammatory phenotype that contributes to chronic obstructive pulmonary disease.

Authors:  Samuel Alam; Zhenjun Li; Carl Atkinson; Danny Jonigk; Sabina Janciauskiene; Ravi Mahadeva
Journal:  Am J Respir Crit Care Med       Date:  2014-04-15       Impact factor: 21.405

4.  Disorders of protein misfolding: alpha-1-antitrypsin deficiency as prototype.

Authors:  Gary A Silverman; Stephen C Pak; David H Perlmutter
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5.  Discovery of an inhibitor of Z-alpha1 antitrypsin polymerization.

Authors:  Valerie Berthelier; Jason Brett Harris; Kasey Noel Estenson; Jerome Baudry
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Review 6.  Mysteries of α1-antitrypsin deficiency: emerging therapeutic strategies for a challenging disease.

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7.  Functional characterization of a SNP (F51S) found in human alpha 1-antitrypsin.

Authors:  Hong-Nhung Trinh; Sei-Heon Jang; ChangWoo Lee
Journal:  Mol Genet Genomic Med       Date:  2019-06-28       Impact factor: 2.183

8.  Gene transfer of master autophagy regulator TFEB results in clearance of toxic protein and correction of hepatic disease in alpha-1-anti-trypsin deficiency.

Authors:  Nunzia Pastore; Keith Blomenkamp; Fabio Annunziata; Pasquale Piccolo; Pratibha Mithbaokar; Rosa Maria Sepe; Francesco Vetrini; Donna Palmer; Philip Ng; Elena Polishchuk; Simona Iacobacci; Roman Polishchuk; Jeffrey Teckman; Andrea Ballabio; Nicola Brunetti-Pierri
Journal:  EMBO Mol Med       Date:  2013-02-04       Impact factor: 12.137

9.  Mineralization of alpha-1-antitrypsin inclusion bodies in Mmalton alpha-1-antitrypsin deficiency.

Authors:  Francesco Callea; Isabella Giovannoni; Paola Francalanci; Renata Boldrini; Gavino Faa; Daniela Medicina; Valerio Nobili; Valeer J Desmet; Kamal Ishak; Kuniaki Seyama; Emanuele Bellacchio
Journal:  Orphanet J Rare Dis       Date:  2018-05-16       Impact factor: 4.123

  9 in total

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