Literature DB >> 22409359

Marked intrafamilial phenotypic variation in a family with SOD1 C111Y mutation.

Akinori Nakamura1, Akiyo Hineno, Kunihiro Yoshida, Yoshiki Sekijima, Naoko Hanaoka-Tachibana, Yo-Ichi Takei, Shinji Ohara, Shu-Ichi Ikeda.   

Abstract

Our objectives were to identify the disease-causing mutation in, and report on the clinical features of, a Japanese family that had coexisting phenotypes of amyotrophic lateral sclerosis and spinal muscular atrophy. The family comprised nine patients (six men and three women). We reviewed their clinical records and performed mutation analysis of the copper/zinc superoxide dismutase (SOD1) gene in some of these patients. The patients either had a rapid (n=7) or an extremely long (n=2) clinical course. The mean age at onset was 39.0±13.7 years (range 20-68 years). The initial symptoms were bulbar palsy (n=2), upper (n=4) or lower (n=2) limb muscle weakness, or leg cramps (n=1). The total disease duration varied widely, ranging from one year to >69 years. We identified a SOD1 C111Y mutation among patients in this family. In conclusion, the family showed a marked intrafamilial phenotypic variation associated with the SOD1 C111Y mutation. Elucidating the biological basis of disease expression in patients with the SOD1 C111Y mutation may provide us with useful information to develop therapeutic approaches and to prevent disease progression.

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Year:  2012        PMID: 22409359     DOI: 10.3109/17482968.2011.656311

Source DB:  PubMed          Journal:  Amyotroph Lateral Scler        ISSN: 1471-180X


  6 in total

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Journal:  Proc Natl Acad Sci U S A       Date:  2014-10-14       Impact factor: 11.205

2.  Immunochemical characterization on pathological oligomers of mutant Cu/Zn-superoxide dismutase in amyotrophic lateral sclerosis.

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3.  Analysis of SOD1 mutations in a Chinese population with amyotrophic lateral sclerosis: a case-control study and literature review.

Authors:  QianQian Wei; QingQing Zhou; YongPing Chen; RuWei Ou; Bei Cao; YaQian Xu; Jing Yang; Hui-Fang Shang
Journal:  Sci Rep       Date:  2017-03-14       Impact factor: 4.379

4.  Wild-type Cu/Zn-superoxide dismutase is misfolded in cerebrospinal fluid of sporadic amyotrophic lateral sclerosis.

Authors:  Eiichi Tokuda; Yo-Ichi Takei; Shinji Ohara; Noriko Fujiwara; Isao Hozumi; Yoshiaki Furukawa
Journal:  Mol Neurodegener       Date:  2019-11-19       Impact factor: 14.195

5.  Disulfide scrambling in superoxide dismutase 1 reduces its cytotoxic effect in cultured cells and promotes protein aggregation.

Authors:  Lina Leinartaitė; Ann-Sofi Johansson
Journal:  PLoS One       Date:  2013-10-15       Impact factor: 3.240

6.  Selective Expression of Osteopontin in ALS-resistant Motor Neurons is a Critical Determinant of Late Phase Neurodegeneration Mediated by Matrix Metalloproteinase-9.

Authors:  Yuta Morisaki; Mamiko Niikura; Mizuho Watanabe; Kosuke Onishi; Shogo Tanabe; Yasuhiro Moriwaki; Takashi Okuda; Shinji Ohara; Shigeo Murayama; Masaki Takao; Sae Uchida; Koji Yamanaka; Hidemi Misawa
Journal:  Sci Rep       Date:  2016-06-06       Impact factor: 4.379

  6 in total

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