Literature DB >> 22398098

Therapeutic drug monitoring of thiopurine metabolites in adult thiopurine tolerant IBD patients on maintenance therapy.

Lennard P L Gilissen1, Dennis R Wong, Leopold G J B Engels, Jörgen Bierau, Jaap A Bakker, Aimée D C Paulussen, Mariëlle J Romberg-Camps, Arnold Stronkhorst, Paul Bus, Laurens P Bos, Piet M Hooymans, Reinhold W Stockbrügger, Cees Neef, Ad A M Masclee.   

Abstract

BACKGROUND AND AIMS: Therapeutic drug monitoring of active metabolites of thiopurines, azathioprine and 6-mercaptopurine, is relatively new. The proposed therapeutic threshold level of the active 6-thioguanine nucleotides (6-TGN) is ≥235 pmol/8×10(8) erythrocytes. The aim of this prospective cross-sectional study was to compare 6-TGN levels in adult thiopurine tolerant IBD patients with an exacerbation with those in remission, and to determine the therapeutic 6-TGN cut-off level.
METHODS: Hundred IBD patients were included. Outcome measures were thiopurine metabolite levels, calculated therapeutic 6-TGN cut-off level, CDAI/CAI scores, thiopurine dose and TPMT enzyme activity.
RESULTS: Forty-one patients had an exacerbation, 59 patients were in remission. In 17% of all patients 6-TGN levels were compatible with non-compliance. The median 6-TGN levels were not significantly different between the exacerbation and remission group (227 versus 263 pmol/8×10(8) erythrocytes, p=0.29). The previous reported therapeutic 6-TGN cut-off level of 235 pmol/8×10(8) erythrocytes was confirmed in this study. Twenty-six of the 41 patients (63%) with active disease had 6-TGN levels below this threshold and 24 of 59 IBD patients (41%) in clinical remission (p=0.04).
CONCLUSIONS: Thiopurine non-compliance occurs frequently both in active and quiescent disease. 6-TGN levels below or above the therapeutic threshold are associated with a significant higher chance of IBD exacerbation and remission, respectively. These data support the role of therapeutic drug monitoring in thiopurine maintenance therapy in IBD to reveal non-compliance or underdosing, and can be used as a practical tool to optimize thiopurine therapy, especially in case of thiopurine non-response.
Copyright © 2011 European Crohn's and Colitis Organisation. Published by Elsevier B.V. All rights reserved.

Entities:  

Mesh:

Substances:

Year:  2012        PMID: 22398098     DOI: 10.1016/j.crohns.2011.12.003

Source DB:  PubMed          Journal:  J Crohns Colitis        ISSN: 1873-9946            Impact factor:   9.071


  26 in total

1.  Prevalence of TPMT polymorphism in Indian patients requiring immunomodulator therapy and its clinical significance.

Authors:  Sandeep Kirit Davavala; Devendra C Desai; Philip Abraham; Tester Ashavaid; Anand Joshi; Tarun Gupta
Journal:  Indian J Gastroenterol       Date:  2013-08-31

2.  The glutathione transferase Mu null genotype leads to lower 6-MMPR levels in patients treated with azathioprine but not with mercaptopurine.

Authors:  M M T J Broekman; D R Wong; G J A Wanten; H M Roelofs; C J van Marrewijk; O H Klungel; A L M Verbeek; P M Hooymans; H-J Guchelaar; H Scheffer; L J J Derijks; M J H Coenen; D J de Jong
Journal:  Pharmacogenomics J       Date:  2017-01-03       Impact factor: 3.550

3.  Oral administration of non-absorbable delayed release 6-mercaptopurine is locally active in the gut, exerts a systemic immune effect and alleviates Crohn's disease with low rate of side effects: results of double blind Phase II clinical trial.

Authors:  E Israeli; E Goldin; S Fishman; F Konikoff; A Lavy; Y Chowers; E Melzer; A Lahat; M Mahamid; H Shirin; E Nussinson; O Segol; A Ben Ya'acov; Y Shabbat; Y Ilan
Journal:  Clin Exp Immunol       Date:  2015-08       Impact factor: 4.330

4.  Measurements of 6-thioguanine nucleotide levels with TPMT and NUDT15 genotyping in patients with Crohn's disease.

Authors:  Ji Hyeon Lee; Tae Jun Kim; Eun Ran Kim; Sung Noh Hong; Dong Kyung Chang; Li-Hwa Choi; Hye In Woo; Soo-Youn Lee; Young-Ho Kim
Journal:  PLoS One       Date:  2017-12-05       Impact factor: 3.240

Review 5.  Use of thiopurines in inflammatory bowel disease: Safety issues.

Authors:  Anastasia Konidari; Wael El Matary
Journal:  World J Gastrointest Pharmacol Ther       Date:  2014-05-06

6.  Metabolite monitoring to guide thiopurine therapy in systemic autoimmune diseases.

Authors:  Aurélie Chapdelaine; Anne-Marie Mansour; Yves Troyanov; David R Williamson; Maxime Doré
Journal:  Clin Rheumatol       Date:  2017-01-27       Impact factor: 2.980

Review 7.  Monitoring thiopurine metabolites in inflammatory bowel disease.

Authors:  Yago González-Lama; Javier P Gisbert
Journal:  Frontline Gastroenterol       Date:  2016-04-07

Review 8.  Optimization of the treatment with immunosuppressants and biologics in inflammatory bowel disease.

Authors:  Sara Renna; Mario Cottone; Ambrogio Orlando
Journal:  World J Gastroenterol       Date:  2014-08-07       Impact factor: 5.742

9.  Do clinical and laboratory parameters predict thiopurine metabolism and clinical outcome in patients with inflammatory bowel diseases?

Authors:  Sven Frick; Daniel Müller; Gerd A Kullak-Ublick; Alexander Jetter
Journal:  Eur J Clin Pharmacol       Date:  2019-01-04       Impact factor: 2.953

Review 10.  Emerging oral targeted therapies in inflammatory bowel diseases: opportunities and challenges.

Authors:  Marcel Vetter; Markus F Neurath
Journal:  Therap Adv Gastroenterol       Date:  2017-09-05       Impact factor: 4.409

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.