Literature DB >> 22369159

Hypocalcemia and hypovitaminosis D in dogs with induced endotoxemia.

M K Holowaychuk1, A J Birkenheuer, J Li, H Marr, A Boll, S K Nordone.   

Abstract

BACKGROUND: Hypocalcemia is a documented electrolyte disturbance in people and animals with sepsis, but its mechanism is poorly understood.
OBJECTIVE: To investigate mechanisms of hypocalcemia in dogs with experimentally induced endotoxemia. ANIMALS: Six healthy mixed breed dogs were included in this nonrandomized, placebo-controlled, crossover study.
METHODS: Dogs initially were injected with placebo (0.9% NaCl; 1 mL, IV) and then lipopolysaccharide (LPS; 2 μg/kg, IV) after a 5-day washout period. Blood and urine samples were collected for measurement of serum total calcium (tCa), ionized calcium (iCa), total magnesium (tMg), ionized magnesium (iMg), parathyroid hormone (PTH), 25-hydroxyvitamin D (vitamin D), venous blood gases, and fractional excretion (FE) of calcium.
RESULTS: After LPS administration, body temperature increased and blood pressure decreased. Both iCa and tCa decreased (P < .01), but iMg was not significantly different between control and LPS treatments. PTH concentrations increased (P < .01) and vitamin D concentrations decreased (P < .01). Venous pH, bicarbonate, base excess, and blood glucose also decreased (P < .01). Urine tCa concentration was below the limit of detection for all dogs after LPS administration.
CONCLUSIONS: Hypocalcemia occurs during endotoxemia in dogs and is associated with hypovitaminosis D. Hypomagnesemia, hypoparathyroidism, alkalosis, and increased calciuresis are not associated with hypocalcemia in endotoxemic dogs.
Copyright © 2012 by the American College of Veterinary Internal Medicine.

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Year:  2012        PMID: 22369159     DOI: 10.1111/j.1939-1676.2012.00886.x

Source DB:  PubMed          Journal:  J Vet Intern Med        ISSN: 0891-6640            Impact factor:   3.333


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