Literature DB >> 22342252

Neuropathic pain: an evolutionary hypothesis.

John C Ashton1.   

Abstract

BACKGROUND: Whereas nociceptive pain has a clear survival value, the evolutionary origins of neuropathic pain remains unexplained.
OBJECTIVES: It is argued that neuropathic pain is an adaptation that has evolved to detect non-specific damage to the nervous system, and that it operates on the same principles of an analogous hypothesis that has been put forward to explain the evolutionary utility of motion sickness. Whereas motion sickness has been proposed to arise from an inappropriate activation of a system evolved to respond to incoherence between vestibular and visual reference frames as an indication of acute neurotoxicity, it is proposed that neuropathic pain arises from the activation of a system evolved to respond to incoherence between proprioceptive and motor outputs as an indication of nerve trauma. RESULTS AND
CONCLUSIONS: Evidence that supports this hypothesis is reviewed, followed by conclusions regarding consequences for pain theory and management.
Copyright © 2012 Elsevier Ltd. All rights reserved.

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Year:  2012        PMID: 22342252     DOI: 10.1016/j.mehy.2012.01.044

Source DB:  PubMed          Journal:  Med Hypotheses        ISSN: 0306-9877            Impact factor:   1.538


  2 in total

1.  Activation of neurotrophins in lumbar dorsal root probably contributes to neuropathic pain after spinal nerve ligation.

Authors:  Abdolreza Kazemi; Masoud Rahmati; Rasoul Eslami; Vahid Sheibani
Journal:  Iran J Basic Med Sci       Date:  2017-01       Impact factor: 2.699

2.  Oxycodone regulates incision-induced activation of neurotrophic factors and receptors in an acute post-surgery pain rat model.

Authors:  Baowen Liu; Yi Liu; Ningbo Li; Jin Zhang; Xianwei Zhang
Journal:  J Pain Res       Date:  2018-10-30       Impact factor: 3.133

  2 in total

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