Literature DB >> 22335230

Thermal sensitivity of endothelial cells on synthetic vascular graft material.

Mark R Brinton1, Chad A Tagge, Russell J Stewart, Alfred K Cheung, Yan-Ting E Shiu, Douglas A Christensen.   

Abstract

PURPOSE: The goal is to identify thermal exposures capable of reducing or eliminating cell survival on expanded polytetrafluoroethylene (ePTFE), in an effort to develop a mild hyperthermia treatment of neointimal hyperplasia in ePTFE vascular grafts.
MATERIALS AND METHODS: Viable and dead bovine aortic endothelial cells were quantified following different thermal exposure conditions: cells on collagen-coated ePTFE sheets or tissue culture polystyrene dishes were heated at 42° and 45°C to determine their thermal sensitivity on different surfaces, and cells cultured on collagen-coated ePTFE sheets were heated at 43-50°C for various durations, followed by incubation at 37°C for 0 and 20 h, respectively. Significant cell death was set to be 50%. Two types of cell death, apoptosis and necrosis, were distinguished by cell morphology and membrane integrity assessments.
RESULTS: The attachment and survival of cells on ePTFE sheets were more sensitive to inhibition by mild heating than those on tissue culture dishes. Exposure to 45°C for 90 min and 50°C for 30 min caused significant necrotic cell death on ePTFE (65% and 75%, respectively). A 37°C/20-h incubation following 30-min exposures at 47° and 50°C increased total cell death (necrosis + apoptosis) from 20% to 50% and 75% to 100%, respectively.
CONCLUSION: Cells grown on ePTFE were more susceptible to mild hyperthermia-induced death, compared to those on tissue culture dishes. Significant cell death on ePTFE mainly via apoptosis can be achieved by optimising temperature and duration of exposure.

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Year:  2012        PMID: 22335230     DOI: 10.3109/02656736.2011.638963

Source DB:  PubMed          Journal:  Int J Hyperthermia        ISSN: 0265-6736            Impact factor:   3.914


  10 in total

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Authors:  Zhi-Feng Liu; Dong Zheng; Guo-Chang Fan; Tianqing Peng; Lei Su
Journal:  Apoptosis       Date:  2016-08       Impact factor: 4.677

2.  Down-regulation of miR-181a can reduce heat stress damage in PBMCs of Holstein cows.

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Journal:  In Vitro Cell Dev Biol Anim       Date:  2016-04-29       Impact factor: 2.416

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Authors:  F Wu; X J Dong; H Q Zhang; L Li; Q L Xu; Z F Liu; Z T Gu; L Su
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4.  Heat stress induced apoptosis is triggered by transcription-independent p53, Ca(2+) dyshomeostasis and the subsequent Bax mitochondrial translocation.

Authors:  Z T Gu; L Li; F Wu; P Zhao; H Yang; Y S Liu; Y Geng; M Zhao; L Su
Journal:  Sci Rep       Date:  2015-06-24       Impact factor: 4.379

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Authors:  Li Li; Hongping Tan; Zhengtao Gu; Zhifeng Liu; Yan Geng; Yunsong Liu; Huasheng Tong; Youqing Tang; Junmin Qiu; Lei Su
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Authors:  Fang Chen; Huimin Li; Guoguo Zhu; Xiaojuan Chen; Zhongzhi Tang
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Authors:  Nancy K Ramadan; Gamal Badr; Hanem S Abdel-Tawab; Samia F Ahmed; Mohamed H Mahmoud
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9.  NF-κB signaling is essential for resistance to heat stress-induced early stage apoptosis in human umbilical vein endothelial cells.

Authors:  Yanan Liu; Gengbiao Zhou; Zhenglian Wang; Xiaohua Guo; Qiulin Xu; Qiaobing Huang; Lei Su
Journal:  Sci Rep       Date:  2015-09-04       Impact factor: 4.379

10.  Heat stress induces apoptosis through transcription-independent p53-mediated mitochondrial pathways in human umbilical vein endothelial cell.

Authors:  Z T Gu; H Wang; L Li; Y S Liu; X B Deng; S F Huo; F F Yuan; Z F Liu; H S Tong; L Su
Journal:  Sci Rep       Date:  2014-03-26       Impact factor: 4.379

  10 in total

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