Literature DB >> 22284962

Lack of periconceptional vitamins or supplements that contain folic acid and diabetes mellitus-associated birth defects.

Adolfo Correa1, Suzanne M Gilboa, Lorenzo D Botto, Cynthia A Moore, Charlotte A Hobbs, Mario A Cleves, Tiffany J Riehle-Colarusso, D Kim Waller, E Albert Reece.   

Abstract

OBJECTIVE: The purpose of this study was to examine the risk of birth defects in relation to diabetes mellitus and the lack of use of periconceptional vitamins or supplements that contain folic acid. STUDY
DESIGN: The National Birth Defects Prevention Study (1997-2004) is a multicenter, population-based case-control study of birth defects (14,721 cases and 5437 control infants). Cases were categorized into 18 types of heart defects and 26 noncardiac birth defects. We estimated odds ratios for independent and joint effects of preexisting diabetes mellitus and a lack of periconceptional use of vitamins or supplements that contain folic acid.
RESULTS: The pattern of odds ratios suggested an increased risk of defects that are associated with diabetes mellitus in the absence vs the presence of the periconceptional use of vitamins or supplements that contain folic acid.
CONCLUSION: The lack of periconceptional use of vitamins or supplements that contain folic acid may be associated with an excess risk for birth defects due to diabetes mellitus. Published by Mosby, Inc.

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Year:  2011        PMID: 22284962      PMCID: PMC4915337          DOI: 10.1016/j.ajog.2011.12.018

Source DB:  PubMed          Journal:  Am J Obstet Gynecol        ISSN: 0002-9378            Impact factor:   8.661


  46 in total

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6.  Diabetes mellitus and birth defects.

Authors:  Adolfo Correa; Suzanne M Gilboa; Lilah M Besser; Lorenzo D Botto; Cynthia A Moore; Charlotte A Hobbs; Mario A Cleves; Tiffany J Riehle-Colarusso; D Kim Waller; E Albert Reece
Journal:  Am J Obstet Gynecol       Date:  2008-07-31       Impact factor: 8.661

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10.  The green tea polyphenol EGCG alleviates maternal diabetes-induced neural tube defects by inhibiting DNA hypermethylation.

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