Literature DB >> 22268491

Middle cerebellar peduncles and Pontine T2 hypointensities in ARSACS.

Haruo Shimazaki1, Yoshihisa Takiyama, Junko Honda, Kumi Sakoe, Michito Namekawa, Jun Tsugawa, Yoshio Tsuboi, Chieko Suzuki, Masayuki Baba, Imaharu Nakano.   

Abstract

BACKGROUND AND
PURPOSE: Magnetic resonance imaging (MRI) of autosomal recessive spastic ataxia of Charlevoix-Saguenay (ARSACS) cases in Quebec and Europe was reported to show linear hypointensities in T2-weighted and Fluid Attenuated Inversion Recovery (FLAIR) images of the pons. We attempted to clarify the characteristics of the brain MRI findings in ARSACS cases.
METHODS: Eight Japanese early-onset ataxia patients with ARSACS confirmed molecularly were investigated. We performed neurological examination, SACS gene analysis, and MRI in the patients.
RESULTS: Hypointensity lesions in the middle cerebellar peduncles in addition to the pons were observed in T2-weighted and FLAIR images in all eight cases. Although superior cerebellar atrophy was seen in all cases, this MRI finding might not be specific for ARSACS. Upper cervical cord and medulla oblongata atrophy was not observed in 3 of the 7 patients examined.
CONCLUSION: Not only pontine but also middle cerebellar peduncle hypointensity lesions observed in T2-weighted and FLAIR images could be specific findings for ARSACS even in cases with variable clinical phenotypes.
Copyright © 2012 by the American Society of Neuroimaging.

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Year:  2012        PMID: 22268491     DOI: 10.1111/j.1552-6569.2011.00647.x

Source DB:  PubMed          Journal:  J Neuroimaging        ISSN: 1051-2284            Impact factor:   2.486


  7 in total

Review 1.  A novel homozygous SACS mutation identified by whole exome sequencing-genotype phenotype correlations of all published cases.

Authors:  Georgia Xiromerisiou; Katerina Dadouli; Chrysoula Marogianni; Antonios Provatas; Panagiotis Ntellas; Dimitrios Rikos; Pantelis Stathis; Despina Georgouli; Gedeon Loules; Maria Zamanakou; Georgios M Hadjigeorgiou
Journal:  J Mol Neurosci       Date:  2019-11-07       Impact factor: 3.444

2.  SACS variants are a relevant cause of autosomal recessive hereditary motor and sensory neuropathy.

Authors:  Katharina Vill; Wolfgang Müller-Felber; Dieter Gläser; Marius Kuhn; Veronika Teusch; Herbert Schreiber; Joachim Weis; Jörg Klepper; Anja Schirmacher; Astrid Blaschek; Manuela Wiessner; Tim M Strom; Bianca Dräger; Kristina Hofmeister-Kiltz; Moritz Tacke; Lucia Gerstl; Peter Young; Rita Horvath; Jan Senderek
Journal:  Hum Genet       Date:  2018-11-21       Impact factor: 4.132

3.  Assessment of whole-brain white matter by DTI in autosomal recessive spastic ataxia of Charlevoix-Saguenay.

Authors:  K K Oguz; G Haliloglu; C Temucin; R Gocmen; A C Has; K Doerschner; A Dolgun; M Alikasifoglu
Journal:  AJNR Am J Neuroradiol       Date:  2013-04-18       Impact factor: 3.825

4.  Complicated paroxysmal kinesigenic dyskinesia associated with SACS mutations.

Authors:  Qiang Lu; Liang Shang; Wo Tu Tian; Li Cao; Xue Zhang; Qing Liu
Journal:  Ann Transl Med       Date:  2020-01

Review 5.  Genetics of Autosomal Recessive Spastic Ataxia of Charlevoix-Saguenay (ARSACS) and Role of Sacsin in Neurodegeneration.

Authors:  Jaya Bagaria; Eva Bagyinszky; Seong Soo A An
Journal:  Int J Mol Sci       Date:  2022-01-04       Impact factor: 5.923

Review 6.  Documenting manifestations and impacts of autosomal recessive spastic ataxia of Charlevoix-Saguenay to develop patient-reported outcome.

Authors:  Marjolaine Tremblay; Laura Girard-Côté; Bernard Brais; Cynthia Gagnon
Journal:  Orphanet J Rare Dis       Date:  2022-10-01       Impact factor: 4.303

7.  Autosomal Recessive Spastic Ataxia of Charlevoix-Saguenay without Spasticity.

Authors:  Izumi Aida; Tetsuo Ozawa; Hidehiko Fujinaka; Kiyoe Goto; Kentaro Ohta; Takashi Nakajima
Journal:  Intern Med       Date:  2021-06-12       Impact factor: 1.271

  7 in total

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