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Literature DB >> 22266724

Differential sensitivity of RIP3-proficient and deficient murine fibroblasts to camptothecin anticancer drugs.

Jin-xue He, Ying-qing Wang, Jian-ming Feng, Jia-xin Li, Lei Xu, Xiao-hua Li, Wei Wang, Xia-juan Huan, Yi Jiang, Bing Yu, Guang Chen, Ze-hong Miao.

Abstract

Entities: Chemical Gene Species

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Year: 2012 PMID： 22266724 PMCID： PMC4077140 DOI： 10.1038/aps.2012.1

Source DB: PubMed Journal: Acta Pharmacol Sin ISSN： 1671-4083 Impact factor: 6.150

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11 in total

1. Multiple death pathways in TNF-treated fibroblasts: RIP3- and RIP1-dependent and independent routes.

Authors: Duan-Wu Zhang; Min Zheng; Jing Zhao; Yuan-Yue Li; Zhe Huang; Zhu Li; Jiahuai Han
Journal: Cell Res Date: 2011-01-04 Impact factor: 25.617

Review 2. Extrinsic versus intrinsic apoptosis pathways in anticancer chemotherapy.

Authors: S Fulda; K-M Debatin
Journal: Oncogene Date: 2006-08-07 Impact factor: 9.867

3. Complementary roles of Fas-associated death domain (FADD) and receptor interacting protein kinase-3 (RIPK3) in T-cell homeostasis and antiviral immunity.

Authors: Jennifer V Lu; Brian M Weist; Bram J van Raam; Brett S Marro; Long V Nguyen; Prathna Srinivas; Bryan D Bell; Keith A Luhrs; Thomas E Lane; Guy S Salvesen; Craig M Walsh
Journal: Proc Natl Acad Sci U S A Date: 2011-08-29 Impact factor: 11.205

Review 4. Molecular mechanisms of necroptosis: an ordered cellular explosion.

Authors: Peter Vandenabeele; Lorenzo Galluzzi; Tom Vanden Berghe; Guido Kroemer
Journal: Nat Rev Mol Cell Biol Date: 2010-09-08 Impact factor: 94.444

5. Chk1 and Chk2 are differentially involved in homologous recombination repair and cell cycle arrest in response to DNA double-strand breaks induced by camptothecins.

Authors: Min Huang; Ze-Hong Miao; Hong Zhu; Yu-Jun Cai; Wei Lu; Jian Ding
Journal: Mol Cancer Ther Date: 2008-06 Impact factor: 6.261

Review 6. Mechanisms of resistance to topoisomerase I-targeting drugs.

Authors: Zeshaan A Rasheed; Eric H Rubin
Journal: Oncogene Date: 2003-10-20 Impact factor: 9.867

7. Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation.

Authors: Young Sik Cho; Sreerupa Challa; David Moquin; Ryan Genga; Tathagat Dutta Ray; Melissa Guildford; Francis Ka-Ming Chan
Journal: Cell Date: 2009-06-12 Impact factor: 41.582

Differential sensitivity of RIP3-proficient and deficient murine fibroblasts to camptothecin anticancer drugs.

1. Multiple death pathways in TNF-treated fibroblasts: RIP3- and RIP1-dependent and independent routes.

Review 2. Extrinsic versus intrinsic apoptosis pathways in anticancer chemotherapy.

3. Complementary roles of Fas-associated death domain (FADD) and receptor interacting protein kinase-3 (RIPK3) in T-cell homeostasis and antiviral immunity.

Review 4. Molecular mechanisms of necroptosis: an ordered cellular explosion.

5. Chk1 and Chk2 are differentially involved in homologous recombination repair and cell cycle arrest in response to DNA double-strand breaks induced by camptothecins.

Review 6. Mechanisms of resistance to topoisomerase I-targeting drugs.

7. Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation.

8. Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha.

9. Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis.

10. RIP3 mediates the embryonic lethality of caspase-8-deficient mice.

1. RIP3 overexpression sensitizes human breast cancer cells to parthenolide in vitro via intracellular ROS accumulation.

2. The B-Raf(V600E) inhibitor dabrafenib selectively inhibits RIP3 and alleviates acetaminophen-induced liver injury.