Literature DB >> 22207710

FRS2α-mediated FGF signals suppress premature differentiation of cardiac stem cells through regulating autophagy activity.

Jue Zhang1, Junchen Liu, Yanqing Huang, Julia Y F Chang, Leyuan Liu, Wallace L McKeehan, James F Martin, Fen Wang.   

Abstract

RATIONALE: Although the fibroblast growth factor (FGF) signaling axis plays important roles in heart development, the molecular mechanism by which the FGF regulates cardiogenesis is not fully understood.
OBJECTIVE: To investigate the mechanism by which FGF signaling regulates cardiac progenitor cell differentiation. METHODS AND
RESULTS: Using mice with tissue-specific ablation of FGF receptors and FGF receptor substrate 2α (Frs2α) in heart progenitor cells, we demonstrate that disruption of FGF signaling leads to premature differentiation of cardiac progenitor cells in mice. Using embryoid body cultures of mouse embryonic stem cells, we reveal that FGF signaling promotes mesoderm differentiation in embryonic stem cells but inhibits cardiomyocyte differentiation of the mesoderm cells at later stages. Furthermore, we also report that inhibiting FRS2α-mediated signals increases autophagy and that activating autophagy promotes myocardial differentiation and vice versa.
CONCLUSIONS: The results indicate that the FGF/FRS2α-mediated signals prevent premature differentiation of heart progenitor cells through suppressing autophagy. The findings provide the first evidence that autophagy plays a role in heart progenitor differentiation.

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Year:  2011        PMID: 22207710      PMCID: PMC3677753          DOI: 10.1161/CIRCRESAHA.111.255950

Source DB:  PubMed          Journal:  Circ Res        ISSN: 0009-7330            Impact factor:   17.367


  56 in total

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Review 10.  Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes.

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Journal:  Autophagy       Date:  2007-11-21       Impact factor: 16.016

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  33 in total

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Review 7.  A time to reap, a time to sow: mitophagy and biogenesis in cardiac pathophysiology.

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8.  FGFR3/fibroblast growth factor receptor 3 inhibits autophagy through decreasing the ATG12-ATG5 conjugate, leading to the delay of cartilage development in achondroplasia.

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