Literature DB >> 22140601

Differentiated intestinal epithelial cells express high levels of TGF-β receptors and exhibit increased sensitivity to growth inhibition.

Navneeta Rathor1, Shelley R Wang, Elizabeth T Chang, Jaladanki N Rao.   

Abstract

BACKGROUND: Intestinal epithelial cells (IECs) within crypts continuously divide and differentiate as they migrate up towards the luminal surface of the mucosa. With the onset of differentiation, IECs lose their proliferative potential, but the exact mechanism remains unknown. This current study examined the involvement of the TGF-β signaling pathway in this process.
METHODS: Studies were conducted in the IEC-6 cell line derived from rat small intestinal crypt cells. Cell differentiation was induced by forced expression of the Cdx2 gene, a transcription factor responsible for controlling intestinal epithelial cell differentiation.
RESULTS: Forced expression of the Cdx2 gene in stable Cdx2-transfected IEC-6 cells resulted in a differentiated phenotype as indicated by morphological features and increased expression of sucrase-isomaltase. Levels of TGF-β type I receptor (TGFβ-RI) and TGF-β type II receptor (TGFβ-RII) increased in these differentiated epithelial cells. The induced TGFβ-RI and TGFβ-RII expression in Cdx2-transfected IEC-6 cells was associated with increased sensitivity to TGF-β-induced growth inhibition. Depletion of cellular polyamines further increased TGF-β receptor expression and additionally enhanced the response to TGF-β-induced growth inhibition. Increased TGFβ-RI and RII in polyamine-deficient cells were also associated with an induction in JunD/AP-1 activity.
CONCLUSIONS: These results indicate that the loss of the proliferative potential in differentiated IECs results partially from the increased expression of TGF-β receptors.

Entities:  

Keywords:  AP-1 binding sites; Cdx2 gene; TGF-β Receptors; cell growth; electrophoretic mobility shift assay; intestinal epithelium; supershift assays

Year:  2011        PMID: 22140601      PMCID: PMC3228585     

Source DB:  PubMed          Journal:  Int J Clin Exp Med        ISSN: 1940-5901


  37 in total

1.  Polyamines are required for phospholipase C-gamma1 expression promoting intestinal epithelial restitution after wounding.

Authors:  Jaladanki N Rao; Lan Liu; Tongtong Zou; Bernard S Marasa; Dessy Boneva; Shelley R Wang; Debra L Malone; Douglas J Turner; Jian-Ying Wang
Journal:  Am J Physiol Gastrointest Liver Physiol       Date:  2006-09-14       Impact factor: 4.052

2.  Cleavage of structural proteins during the assembly of the head of bacteriophage T4.

Authors:  U K Laemmli
Journal:  Nature       Date:  1970-08-15       Impact factor: 49.962

3.  Inhibition of polyamine synthesis induces p53 gene expression but not apoptosis.

Authors:  L Li; J Li; J N Rao; M Li; B L Bass; J Y Wang
Journal:  Am J Physiol       Date:  1999-04

Review 4.  TGF-beta and fibrosis in different organs - molecular pathway imprints.

Authors:  Dirk Pohlers; Julia Brenmoehl; Ivonne Löffler; Cornelia K Müller; Carola Leipner; Stefan Schultze-Mosgau; Andreas Stallmach; Raimund W Kinne; Gunter Wolf
Journal:  Biochim Biophys Acta       Date:  2009-06-17

5.  Role of receptor complexes in resistance or sensitivity to growth inhibition by TGF beta in intestinal epithelial cell clones.

Authors:  K M Mulder; P R Segarini; S L Morris; J M Ziman; H G Choi
Journal:  J Cell Physiol       Date:  1993-01       Impact factor: 6.384

6.  TGF-beta receptor inactivation and mutant Kras induce intestinal neoplasms in mice via a beta-catenin-independent pathway.

Authors:  Patty Trobridge; Sue Knoblaugh; M Kay Washington; Nina M Munoz; Karen D Tsuchiya; Andres Rojas; Xiaoling Song; Cornelia M Ulrich; Takehiko Sasazuki; Senji Shirasawa; William M Grady
Journal:  Gastroenterology       Date:  2009-02-04       Impact factor: 22.682

Review 7.  Smad3: a key player in pathogenetic mechanisms dependent on TGF-beta.

Authors:  Anita B Roberts; Angelo Russo; Angelina Felici; Kathleen C Flanders
Journal:  Ann N Y Acad Sci       Date:  2003-05       Impact factor: 5.691

8.  Ornithine decarboxylase is important in intestinal mucosal maturation and recovery from injury in rats.

Authors:  G D Lux; L J Marton; S B Baylin
Journal:  Science       Date:  1980-10-10       Impact factor: 47.728

9.  JunD stabilization results in inhibition of normal intestinal epithelial cell growth through P21 after polyamine depletion.

Authors:  Li Li; Lan Liu; Jaladanki N Rao; Ali Esmaili; Eric D Strauch; Barbara L Bass; Jian-Ying Wang
Journal:  Gastroenterology       Date:  2002-09       Impact factor: 22.682

10.  Epithelioid cell cultures from rat small intestine. Characterization by morphologic and immunologic criteria.

Authors:  A Quaroni; J Wands; R L Trelstad; K J Isselbacher
Journal:  J Cell Biol       Date:  1979-02       Impact factor: 10.539

View more
  3 in total

1.  Polyamines regulate intestinal epithelial restitution through TRPC1-mediated Ca²+ signaling by differentially modulating STIM1 and STIM2.

Authors:  Jaladanki N Rao; Navneeta Rathor; Ran Zhuang; Tongtong Zou; Lan Liu; Lan Xiao; Douglas J Turner; Jian-Ying Wang
Journal:  Am J Physiol Cell Physiol       Date:  2012-05-16       Impact factor: 4.249

2.  Polyamines and Gut Mucosal Homeostasis.

Authors:  Jennifer Timmons; Elizabeth T Chang; Jian-Ying Wang; Jaladanki N Rao
Journal:  J Gastrointest Dig Syst       Date:  2012-02-20

3.  Induced PDK1 kinase activity suppresses apoptosis in intestinal epithelial cells by activating Akt signaling following polyamine depletion.

Authors:  Kaspar M Keledjian; Bernard S Marasa; Jian-Ying Wang; Jaladanki N Rao
Journal:  Int J Clin Exp Med       Date:  2012-06-15
  3 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.