Literature DB >> 22131170

Transient locked-in syndrome and basilar artery vasospasm.

G Lacroix1, D Couret, X Combaz, B Prunet, N Girard, N Bruder.   

Abstract

BACKGROUND: Cerebral vasospasm is the main cause of neurological mortality and morbidity following subarachnoid hemorrhage. Basilar artery vasospasm (BAVS) is associated with a high morbidity and may have multiple clinical presentations.
METHODS: We report the case of a 43 years-old man with BAVS presenting as a reversible locked-in syndrome (LIS) after stopping sedation.
RESULTS: The symptoms were successfully managed by intra-arterial infusion of vasodilators and balloon angioplasty. Magnetic resonance imaging did not reveal any brainstem lesion 48 h after the complication, demonstrating a hemodynamic mechanism.
CONCLUSION: LIS can reveal BAVS. Its diagnosis relies on clinical examination. In this case, rapid neuro-interventional treatment permitted reversal of symptoms. This could not have been possible under sedation.

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Year:  2012        PMID: 22131170     DOI: 10.1007/s12028-011-9655-z

Source DB:  PubMed          Journal:  Neurocrit Care        ISSN: 1541-6933            Impact factor:   3.210


  9 in total

Review 1.  CT perfusion cerebral blood flow imaging in neurological critical care.

Authors:  Mark R Harrigan; Jody Leonardo; Kevin J Gibbons; Lee R Guterman; L Nelson Hopkins
Journal:  Neurocrit Care       Date:  2005       Impact factor: 3.210

2.  Transcranial Doppler grading criteria for basilar artery vasospasm.

Authors:  Gill E Sviri; Basavaraj Ghodke; Gavin W Britz; Colleen M Douville; David R Haynor; Ali H Mesiwala; Arthur M Lam; David W Newell
Journal:  Neurosurgery       Date:  2006-08       Impact factor: 4.654

3.  How does care differ for neurological patients admitted to a neurocritical care unit versus a general ICU?

Authors:  Pedro Kurtz; Vincent Fitts; Zeynep Sumer; Hillary Jalon; Joseph Cooke; Vladimir Kvetan; Stephan A Mayer
Journal:  Neurocrit Care       Date:  2011-12       Impact factor: 3.210

4.  Hemodynamic consequences of cerebral vasospasm on perforating arteries: a phantom model study.

Authors:  J F Soustiel; E Levy; R Bibi; S Lukaschuk; D Manor
Journal:  Stroke       Date:  2001-03       Impact factor: 7.914

5.  Peduncular hallucinations following subarachnoid haemorrhage.

Authors:  S B O'Neill; B Pentland; R Sellar
Journal:  Br J Neurosurg       Date:  2005-08       Impact factor: 1.596

6.  Impact of basilar artery vasospasm on outcome in patients with severe cerebral vasospasm after aneurysmal subarachnoid hemorrhage.

Authors:  Gill E Sviri; David W Newell; David H Lewis; Colleen Douville; Basavaraj Ghodke; Minku Chowdhary; Arthur M Lam; David Haynor; Menashe Zaaroor; Gavin W Britz
Journal:  Stroke       Date:  2006-09-28       Impact factor: 7.914

7.  Transdermal nicotine replacement therapy in cigarette smokers with acute subarachnoid hemorrhage.

Authors:  David B Seder; J Michael Schmidt; Neeraj Badjatia; Luis Fernandez; Fred Rincon; Jan Claassen; Errol Gordon; Emmanuel Carrera; Pedro Kurtz; Kiwon Lee; E Sander Connolly; Stephan A Mayer
Journal:  Neurocrit Care       Date:  2011-02       Impact factor: 3.210

8.  Medical complications of aneurysmal subarachnoid hemorrhage: a report of the multicenter, cooperative aneurysm study. Participants of the Multicenter Cooperative Aneurysm Study.

Authors:  N J Solenski; E C Haley; N F Kassell; G Kongable; T Germanson; L Truskowski; J C Torner
Journal:  Crit Care Med       Date:  1995-06       Impact factor: 7.598

9.  Delayed cerebral ischemia manifesting as peduncular hallucinosis after aneurysmal subarachnoid hemorrhage--three case reports.

Authors:  K Yano; T Kuroda; Y Tanabe; H Yamada
Journal:  Neurol Med Chir (Tokyo)       Date:  1994-09       Impact factor: 1.742

  9 in total

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