Literature DB >> 22112286

Diabetes treatment and progression of benign prostatic hyperplasia in community-dwelling black and white men.

Aruna V Sarma1, Jennifer L St Sauver, John M Hollingsworth, Debra J Jacobson, Michaela E McGree, Rodney L Dunn, Michael M Lieber, Steven J Jacobsen.   

Abstract

OBJECTIVE: To conduct a study to determine whether diabetes treatment is associated with benign prostatic hyperplasia (BPH)/lower urinary tract symptoms (LUTS) and progression in black and white men. Diabetes has been associated with BPH and LUTS in aging men.
METHODS: Using the Olmsted County Study of Urinary Symptoms and Health Status among Men and the Flint Men's Health Study, we examined how the use of medical therapy (eg, insulin regimens, oral hypoglycemics) related to changes in LUTS severity, maximal urinary flow rate measured by uroflowmetry, prostate volume determined by transrectal ultrasonography, and serum prostate-specific antigen concentrations.
RESULTS: Of the 2226 men participating in the Olmsted County Study of Urinary Symptoms and Health Status among Men and the Flint Men's Health Study, 186 men reported a history of diabetes, 76.9% of whom were treated with medical therapy. Overall, the men with diabetes had significantly greater odds of moderate/severe LUTS (age- and race-adjusted odds ratio 1.37, 95% confidence interval 1.00-1.87) compared with those without diabetes. However, among the diabetic men, those not taking medication had greater odds of moderate/severe LUTS than those taking medication. This association among men not taking medication was seen for 5 of the 7 individual symptoms. The prostate volume and prostate-specific antigen level were not significantly associated with diabetes treatment. No significant differences were observed for the annual change in BPH characteristics by diabetes treatment status.
CONCLUSION: These findings suggest that the presence of diabetes and subsequent poor glycemic control might be less related to prostate growth and more to the dynamic components of lower urinary tract function. Additional evaluations of the associations between glycemic control and BPH progression are warranted.
Copyright © 2012 Elsevier Inc. All rights reserved.

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Year:  2011        PMID: 22112286      PMCID: PMC3341847          DOI: 10.1016/j.urology.2011.08.065

Source DB:  PubMed          Journal:  Urology        ISSN: 0090-4295            Impact factor:   2.649


  29 in total

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2.  Insulin-like growth factor-1, insulin-like growth factor binding protein-3, and body mass index: clinical correlates of prostate volume among Black men.

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3.  Effect of diabetes on lower urinary tract symptoms in patients with benign prostatic hyperplasia.

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5.  Risk factors for clinical benign prostatic hyperplasia in a community-based population of healthy aging men.

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2.  Hyperglycemia, hyperinsulinemia, insulin resistance, and the risk of BPH/LUTS severity and progression over time in community dwelling black men: the Flint Men's Health Study.

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Review 3.  Hyperglycemia and insulin resistance and the risk of BPH/LUTS: an update of recent literature.

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6.  Genome-wide association study identifies a role for the progesterone receptor in benign prostatic hyperplasia risk.

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7.  Benign prostatic hyperplasia is a significant risk factor for bladder cancer in diabetic patients: a population-based cohort study using the National Health Insurance in Taiwan.

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8.  Impact of type 2 diabetes on lower urinary tract symptoms in men: a cohort study.

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9.  Epidemiology and etiology of benign prostatic hyperplasia and bladder outlet obstruction.

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10.  Obstructive voiding symptoms following stereotactic body radiation therapy for prostate cancer.

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