Literature DB >> 22105900

ARK5 is associated with the invasive and metastatic potential of human breast cancer cells.

Xin-Zhong Chang1, Jie Yu, Hai-Yin Liu, Rui-Hua Dong, Xu-Chen Cao.   

Abstract

PURPOSE: To investigate the effects of Akt/ARK5 pathways on the metastatic potential of human breast cancer cells.
MATERIALS AND METHODS: The human ARK5 gene was transfected into MDA-MB-231 cells. Effects of ARK5 on MDA-MB-231 cells were investigated in vitro. The tumorigenicity and spontaneously metastatic capability regulated by ARK5 were determined using an orthotopic xenograft tumor model.
RESULTS: ARK5 enhanced the invasive and metastatic potential of MDA-MB-231 cells under regulation by Akt. The enhancement was associated with increasing MMP-2, MMP-9, and MT1-MMP expression. The results were further demonstrated by RNA interference experiment. In an in vivo study, we also demonstrated that ARK5-transfected breast cancer cells grew faster and had more pulmonary metastases than its parental counterparts.
CONCLUSION: ARK5 led to a more invasive phenotype and metastatic potential in human breast cancer dependent on Akt.

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Year:  2011        PMID: 22105900     DOI: 10.1007/s00432-011-1102-1

Source DB:  PubMed          Journal:  J Cancer Res Clin Oncol        ISSN: 0171-5216            Impact factor:   4.553


  21 in total

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4.  Strong association of ARK5 with tumor invasion and metastasis.

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5.  Regulation of caspase-6 and FLIP by the AMPK family member ARK5.

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  18 in total

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9.  Rewiring of an epithelial differentiation factor, miR-203, to inhibit human squamous cell carcinoma metastasis.

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10.  microRNA-203 suppresses invasion and epithelial-mesenchymal transition induction via targeting NUAK1 in head and neck cancer.

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