Literature DB >> 22103986

Differential expression of the immunoinflammatory response in trauma patients: burn vs. non-burn.

James E Mace1, Myung S Park, Alejandra G Mora, Kevin K Chung, Wenjun Martini, Christopher E White, John B Holcomb, Gerald A Merrill, Michael A Dubick, Steven E Wolf, Charles E Wade, Martin G Schwacha.   

Abstract

RATIONALE: Cytokines are central mediators of the immune-inflammatory response to injury and subsequent multiple organ dysfunction syndrome (MODS). Although previous studies evaluated cytokine levels after trauma, differences between patients with burn and non-burn trauma have not been assessed systematically.
METHODS: A prospective database of trauma patients admitted between May 2004 and September 2007 to the burn or surgical intensive care units within 24 h of injury with an anticipated stay of at least 72 h was analyzed. Sequential clinical and laboratory parameters were collected in the first week, including multiplex analysis data for plasma levels of inflammatory cytokines (IL-6, and IL-8). Patients with known pre-injury coagulopathy were excluded. A Marshall score of 10 or greater was defined as MODS.
RESULTS: A total of 179 patients were enrolled (67 burn and 112 non-burn). Plasma IL-6 and IL-8 levels were markedly elevated in both burn and non-burn patients compared to healthy volunteers. Burn subjects had higher levels of IL-6 and IL-8 than the non-burn on days 1 through 7 after injury. Subjects with burns and at least 30% total body surface area were older and had a lower injury severity score, a higher prevalence of MODS, and correspondingly higher mortality. Multivariate analysis of injury type, MODS, and time did not demonstrate an influence of MODS.
CONCLUSIONS: Burns were associated with a greater and more sustained immune-inflammatory response than non-burn trauma as evidenced by elevated plasma IL-6 and IL-8 levels during the first week. There was no association between MODS and plasma cytokine levels. Published by Elsevier Ltd.

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Year:  2011        PMID: 22103986      PMCID: PMC9479166          DOI: 10.1016/j.burns.2011.10.013

Source DB:  PubMed          Journal:  Burns        ISSN: 0305-4179            Impact factor:   2.609


  33 in total

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