Literature DB >> 22077279

Accelerated hematopoietic toxicity by high energy (56)Fe radiation.

Kamal Datta1, Shubhankar Suman, Daniela Trani, Kathryn Doiron, Jimmy A Rotolo, Bhaskar V S Kallakury, Richard Kolesnick, Michael F Cole, Albert J Fornace.   

Abstract

PURPOSE: There is little information on the relative toxicity of highly charged (Z) high-energy (HZE) radiation in animal models compared to γ or X-rays, and the general assumption based on in vitro studies has been that acute toxicity is substantially greater.
METHODS: C57BL/6J mice were irradiated with (56)Fe ions (1 GeV/nucleon), and acute (within 30 d) toxicity compared to that of γ rays or protons (1 GeV). To assess relative hematopoietic and gastrointestinal toxicity, the effects of (56)Fe ions were compared to γ rays using complete blood count (CBC), bone marrow granulocyte-macrophage colony forming unit (GM-CFU), terminal deoxynucleotidyl transferase dUTP nick end labeling (TUNEL) assay for apoptosis in bone marrow, and intestinal crypt survival.
RESULTS: Although onset was more rapid, (56)Fe ions were only slightly more toxic than γ rays or protons with lethal dose (LD)(50/30) (a radiation dose at which 50% lethality occurs at 30-day) values of 5.8, 7.25, and 6.8 Gy, respectively, with relative biologic effectiveness for (56)Fe ions of 1.25 and 1.06 for protons.
CONCLUSIONS: (56)Fe radiation caused accelerated and more severe hematopoietic toxicity. Early mortality correlated with more profound leukopenia and subsequent sepsis. Results indicate that there is selective enhanced toxicity to bone marrow progenitor cells, which are typically resistant to γ rays, and bone marrow stem cells, because intestinal crypt cells did not show increased HZE toxicity.

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Year:  2011        PMID: 22077279      PMCID: PMC3580183          DOI: 10.3109/09553002.2012.639434

Source DB:  PubMed          Journal:  Int J Radiat Biol        ISSN: 0955-3002            Impact factor:   2.694


  26 in total

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Review 8.  Management of postirradiation infection: lessons learned from animal models.

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9.  Dual effect of p53 on radiation sensitivity in vivo: p53 promotes hematopoietic injury, but protects from gastro-intestinal syndrome in mice.

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Authors:  R E Millard; N M Blackett
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3.  Acute and fractionated exposure to high-LET (56)Fe HZE-particle radiation both result in similar long-term deficits in adult hippocampal neurogenesis.

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5.  An Integrated Multi-Omic Approach to Assess Radiation Injury on the Host-Microbiome Axis.

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10.  Long-term differential changes in mouse intestinal metabolomics after γ and heavy ion radiation exposure.

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