Literature DB >> 22064956

Diversity of myocardial interstitial proteolytic pathways: gene deletion reveals unexpected consequences.

Francis G Spinale.   

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Year:  2011        PMID： 22064956      PMCID： PMC3229095          DOI： 10.1161/CIRCULATIONAHA.111.064071

Source DB:  PubMed          Journal:  Circulation        ISSN： 0009-7322            Impact factor:   29.690


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  11 in total

1.  Cardiac restricted overexpression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarction.

Authors:  Francis G Spinale; Rupak Mukherjee; Juozas A Zavadzkas; Christine N Koval; Shenikqua Bouges; Robert E Stroud; Lawrence W Dobrucki; Albert J Sinusas
Journal:  J Biol Chem       Date:  2010-07-19       Impact factor: 5.157

Review 2.  The tissue inhibitors of metalloproteinases (TIMPs): an ancient family with structural and functional diversity.

Authors:  Keith Brew; Hideaki Nagase
Journal:  Biochim Biophys Acta       Date:  2010-01-15

3.  Effects of selective matrix metalloproteinase inhibitor (PG-116800) to prevent ventricular remodeling after myocardial infarction: results of the PREMIER (Prevention of Myocardial Infarction Early Remodeling) trial.

Authors:  Michael P Hudson; Paul W Armstrong; Witold Ruzyllo; Jose Brum; Lisa Cusmano; Piotr Krzeski; Robert Lyon; Miguel Quinones; Pierre Theroux; Diana Sydlowski; Henry E Kim; Mario J Garcia; Wael A Jaber; W Douglas Weaver
Journal:  J Am Coll Cardiol       Date:  2006-06-21       Impact factor: 24.094

4.  Reduced degradation of the chemokine MCP-3 by matrix metalloproteinase-2 exacerbates myocardial inflammation in experimental viral cardiomyopathy.

Authors:  Dirk Westermann; Kostantinos Savvatis; Diana Lindner; Christin Zietsch; Peter Moritz Becher; Elke Hammer; Markus M Heimesaat; Stefan Bereswill; Uwe Völker; Felicitas Escher; Alexander Riad; Johanna Plendl; Karin Klingel; Wolfgang Poller; Heinz-Peter Schultheiss; Carsten Tschöpe
Journal:  Circulation       Date:  2011-10-10       Impact factor: 29.690

5.  Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress.

Authors:  Vijay Kandalam; Ratnadeep Basu; Linn Moore; Dong Fan; Xiuhua Wang; Diane M Jaworski; Gavin Y Oudit; Zamaneh Kassiri
Journal:  Circulation       Date:  2011-10-10       Impact factor: 29.690

Review 6.  Myocardial remodeling: cellular and extracellular events and targets.

Authors:  Jennifer A Dixon; Francis G Spinale
Journal:  Annu Rev Physiol       Date:  2011       Impact factor: 19.318

7.  A matrix metalloproteinase induction/activation system exists in the human left ventricular myocardium and is upregulated in heart failure.

Authors:  F G Spinale; M L Coker; L J Heung; B R Bond; H R Gunasinghe; T Etoh; A T Goldberg; J L Zellner; A J Crumbley
Journal:  Circulation       Date:  2000-10-17       Impact factor: 29.690

8.  Macrophage-specific metalloelastase (MMP-12) truncates and inactivates ELR+ CXC chemokines and generates CCL2, -7, -8, and -13 antagonists: potential role of the macrophage in terminating polymorphonuclear leukocyte influx.

Authors:  Richard A Dean; Jennifer H Cox; Caroline L Bellac; Alain Doucet; Amanda E Starr; Christopher M Overall
Journal:  Blood       Date:  2008-07-25       Impact factor: 22.113

Review 9.  Myocardial matrix remodeling and the matrix metalloproteinases: influence on cardiac form and function.

Authors:  Francis G Spinale
Journal:  Physiol Rev       Date:  2007-10       Impact factor: 37.312

Review 10.  Matrix metalloproteinases: what do they not do? New substrates and biological roles identified by murine models and proteomics.

Authors:  David Rodríguez; Charlotte J Morrison; Christopher M Overall
Journal:  Biochim Biophys Acta       Date:  2009-10-01
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  4 in total

Review 1.  The history of matrix metalloproteinases: milestones, myths, and misperceptions.

Authors:  Rugmani Padmanabhan Iyer; Nicolle L Patterson; Gregg B Fields; Merry L Lindsey
Journal:  Am J Physiol Heart Circ Physiol       Date:  2012-08-17       Impact factor: 4.733

2.  Differential patterns of replacement and reactive fibrosis in pressure and volume overload are related to the propensity for ischaemia and involve resistin.

Authors:  Elie R Chemaly; Soojeong Kang; Shihong Zhang; LaTronya McCollum; Jiqiu Chen; Ludovic Bénard; K-Raman Purushothaman; Roger J Hajjar; Djamel Lebeche
Journal:  J Physiol       Date:  2013-09-09       Impact factor: 5.182

3.  Increased systemic Th17 cytokines are associated with diastolic dysfunction in children and adolescents with diabetic ketoacidosis.

Authors:  William H Hoffman; Gregory G Passmore; David W Hannon; Monica V Talor; Pam Fox; Catherine Brailer; Dynita Haislip; Cynthia Keel; Glenn Harris; Noel R Rose; Irma Fiordalisi; Daniela Čiháková
Journal:  PLoS One       Date:  2013-08-27       Impact factor: 3.240

4.  Angiogenesis, cardiomyocyte proliferation and anti-fibrotic effects underlie structural preservation post-infarction by intramyocardially-injected cardiospheres.

Authors:  Eleni Tseliou; Geoffrey de Couto; John Terrovitis; Baiming Sun; Liu Weixin; Linda Marbán; Eduardo Marbán
Journal:  PLoS One       Date:  2014-02-18       Impact factor: 3.240
  4 in total

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