Literature DB >> 22049207

This, too, shall pass--like a kidney stone: a possible path to prophylaxis of nephrolithiasis? Focus on "Cholinergic signaling inhibits oxalate transport by human intestinal T84 cells".

John F Heneghan, Seth L Alper.   

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Year:  2011        PMID： 22049207     DOI： 10.1152/ajpcell.00389.2011

Source DB:  PubMed          Journal:  Am J Physiol Cell Physiol        ISSN： 0363-6143            Impact factor:   4.249


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  5 in total

1.  Cholinergic signaling inhibits oxalate transport by human intestinal T84 cells.

Authors:  Hatim A Hassan; Ming Cheng; Peter S Aronson
Journal:  Am J Physiol Cell Physiol       Date:  2011-09-28       Impact factor: 4.249

2.  Induction of enteric oxalate secretion by Oxalobacter formigenes in mice does not require the presence of either apical oxalate transport proteins Slc26A3 or Slc26A6.

Authors:  Marguerite Hatch
Journal:  Urolithiasis       Date:  2019-06-14       Impact factor: 3.436

3.  Sat1 is dispensable for active oxalate secretion in mouse duodenum.

Authors:  Narae Ko; Felix Knauf; Zhirong Jiang; Daniel Markovich; Peter S Aronson
Journal:  Am J Physiol Cell Physiol       Date:  2012-04-18       Impact factor: 4.249

Review 4.  The role of intestinal oxalate transport in hyperoxaluria and the formation of kidney stones in animals and man.

Authors:  Jonathan M Whittamore; Marguerite Hatch
Journal:  Urolithiasis       Date:  2016-12-02       Impact factor: 3.436

5.  Absence of the sulfate transporter SAT-1 has no impact on oxalate handling by mouse intestine and does not cause hyperoxaluria or hyperoxalemia.

Authors:  Jonathan M Whittamore; Christine E Stephens; Marguerite Hatch
Journal:  Am J Physiol Gastrointest Liver Physiol       Date:  2018-11-01       Impact factor: 4.052
  5 in total

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