Literature DB >> 22024061

Smad4 may help to identify a subset of colorectal cancer patients with early recurrence after curative therapy.

Byung Kyu Ahn1, Si-Hyong Jang, Seung Sam Paik, Kang Hong Lee.   

Abstract

BACKGROUND/AIMS: Loss of Smad4 function is associated with the acquisition of advanced colorectal cancer phenotypes. We investigated the role of Smad4 as a prognostic marker after curative therapy.
METHODOLOGY: Four hundred and twenty nine consecutive colorectal cancers were analyzed by tissue microarray-based immunohistochemical assay.
RESULTS: Smad4 protein was expressed in 61.5% (24/39), 53.1% (77/145), 41.3% (78/189) and 34.8% (16/46) of stage I, II, III and IV cancers, respectively. Lymphovascular invasion and lymph node metastasis were strongly correlated with the loss of Smad4 expression (p<0.0001 and p=0.002, respectively). Disease-free survival did not differ between Smad4-positive and Smad4-negative cancers. In stage III disease, time to recurrence after curative therapy was shorter in the Smad4-negative than in the Smad4- positive cancers (20.1±15.1 vs. 34.6 ± 34.1 months, p=0.035).
CONCLUSIONS: Smad4 protein is of no value in predicting recurrence after curative therapy in colorectal cancer, but it may be helpful in identifying a subset of patients with early recurrence after curative therapy.

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Year:  2011        PMID: 22024061     DOI: 10.5754/hge11186

Source DB:  PubMed          Journal:  Hepatogastroenterology        ISSN: 0172-6390


  7 in total

1.  Loss of Smad4 in colorectal cancer induces resistance to 5-fluorouracil through activating Akt pathway.

Authors:  B Zhang; B Zhang; X Chen; S Bae; K Singh; M K Washington; P K Datta
Journal:  Br J Cancer       Date:  2014-01-02       Impact factor: 7.640

2.  A Meta-Analysis of SMAD4 Immunohistochemistry as a Prognostic Marker in Colorectal Cancer.

Authors:  Philip W Voorneveld; Rutger J Jacobs; Liudmila L Kodach; James C H Hardwick
Journal:  Transl Oncol       Date:  2015-02       Impact factor: 4.243

3.  Progression inference for somatic mutations in cancer.

Authors:  Leif E Peterson; Tatiana Kovyrshina
Journal:  Heliyon       Date:  2017-04-11

4.  Loss of TGFβ signaling promotes colon cancer progression and tumor-associated inflammation.

Authors:  Daniel R Principe; Brian DeCant; Jonas Staudacher; Dominic Vitello; Riley J Mangan; Elizabeth A Wayne; Emman Mascariñas; Andrew M Diaz; Jessica Bauer; Ronald D McKinney; Khashayarsha Khazaie; Boris Pasche; David W Dawson; Hidayatullah G Munshi; Paul J Grippo; Barbara Jung
Journal:  Oncotarget       Date:  2017-01-17

5.  Prognostic value of loss of heterozygosity and sub-cellular localization of SMAD4 varies with tumor stage in colorectal cancer.

Authors:  Xu Jia; Chandrakumar Shanmugam; Ravi K Paluri; Nirag C Jhala; Michael P Behring; Venkat R Katkoori; Shajan P Sugandha; Sejong Bae; Temesgen Samuel; Upender Manne
Journal:  Oncotarget       Date:  2017-03-21

6.  Clinicopathological Characterization and Prognostic Implication of SMAD4 Expression in Colorectal Carcinoma.

Authors:  Seung-Yeon Yoo; Ji-Ae Lee; Yunjoo Shin; Nam-Yun Cho; Jeong Mo Bae; Gyeong Hoon Kang
Journal:  J Pathol Transl Med       Date:  2019-06-24

7.  Presence of Concurrent TP53 Mutations Is Necessary to Predict Poor Outcomes within the SMAD4 Mutated Subgroup of Metastatic Colorectal Cancer.

Authors:  Chongkai Wang; Jaideep Sandhu; Amber Tsao; Marwan Fakih
Journal:  Cancers (Basel)       Date:  2022-07-27       Impact factor: 6.575

  7 in total

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