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Literature DB >> 22021135

Skeletal anabolism, PTH, and the bone-vascular axis.

Dwight A Towler¹.

Abstract

Entities: Chemical Disease Gene Species

Mesh：

Substances：

Year: 2011 PMID： 22021135 PMCID： PMC3391981 DOI： 10.1002/jbmr.523

Source DB: PubMed Journal: J Bone Miner Res ISSN： 0884-0431 Impact factor: 6.741

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40 in total

1. Activation of vascular smooth muscle parathyroid hormone receptor inhibits Wnt/beta-catenin signaling and aortic fibrosis in diabetic arteriosclerosis.

Authors: Su-Li Cheng; Jian-Su Shao; Linda R Halstead; Kathryn Distelhorst; Oscar Sierra; Dwight A Towler
Journal: Circ Res Date: 2010-05-20 Impact factor: 17.367

Review 2. The bone-vascular axis in chronic kidney disease.

Authors: Linda Demer; Yin Tintut
Journal: Curr Opin Nephrol Hypertens Date: 2010-07 Impact factor: 2.894

Review 3. Structure-function analysis of VEGF receptor activation and the role of coreceptors in angiogenic signaling.

Authors: Felix S Grünewald; Andrea E Prota; Alexandra Giese; Kurt Ballmer-Hofer
Journal: Biochim Biophys Acta Date: 2009-09-15

4. N-terminal parathyroid hormone-related peptide hyperpolarizes endothelial cells and causes a reduction of the coronary resistance of the rat heart via endothelial hyperpolarization.

Authors: Yaser Abdallah; Günter Ross; Alexandra Dolf; Marcus P Heinemann; Klaus-Dieter Schlüter
Journal: Peptides Date: 2006-06-27 Impact factor: 3.750

5. Osteoblast precursors, but not mature osteoblasts, move into developing and fractured bones along with invading blood vessels.

Authors: Christa Maes; Tatsuya Kobayashi; Martin K Selig; Sophie Torrekens; Sanford I Roth; Susan Mackem; Geert Carmeliet; Henry M Kronenberg
Journal: Dev Cell Date: 2010-08-17 Impact factor: 12.270

6. CXCR2 and CXCR4 antagonistically regulate neutrophil trafficking from murine bone marrow.

Authors: Kyle J Eash; Adam M Greenbaum; Priya K Gopalan; Daniel C Link
Journal: J Clin Invest Date: 2010-07 Impact factor: 14.808

7. Increased skeletal VEGF enhances beta-catenin activity and results in excessively ossified bones.

Authors: Christa Maes; Steven Goossens; Sonia Bartunkova; Benjamin Drogat; Lieve Coenegrachts; Ingrid Stockmans; Karen Moermans; Omar Nyabi; Katharina Haigh; Michael Naessens; Lieven Haenebalcke; Jan P Tuckermann; Marc Tjwa; Peter Carmeliet; Vice Mandic; Jean-Pierre David; Axel Behrens; Andras Nagy; Geert Carmeliet; Jody J Haigh
Journal: EMBO J Date: 2009-12-10 Impact factor: 11.598

8. Endogenous glucocorticoids decrease skeletal angiogenesis, vascularity, hydration, and strength in aged mice.

Authors: Robert S Weinstein; Chao Wan; Qinglan Liu; Ying Wang; Maria Almeida; Charles A O'Brien; Jeff Thostenson; Paula K Roberson; Adele L Boskey; Thomas L Clemens; Stavros C Manolagas
Journal: Aging Cell Date: 2009-12-28 Impact factor: 9.304

9. Continuous elevation of PTH increases the number of osteoblasts via both osteoclast-dependent and -independent mechanisms.

Authors: Robert L Jilka; Charles A O'Brien; Shoshana M Bartell; Robert S Weinstein; Stavros C Manolagas
Journal: J Bone Miner Res Date: 2010-11 Impact factor: 6.741

Skeletal anabolism, PTH, and the bone-vascular axis.

1. Activation of vascular smooth muscle parathyroid hormone receptor inhibits Wnt/beta-catenin signaling and aortic fibrosis in diabetic arteriosclerosis.

Review 2. The bone-vascular axis in chronic kidney disease.

Review 3. Structure-function analysis of VEGF receptor activation and the role of coreceptors in angiogenic signaling.

4. N-terminal parathyroid hormone-related peptide hyperpolarizes endothelial cells and causes a reduction of the coronary resistance of the rat heart via endothelial hyperpolarization.

5. Osteoblast precursors, but not mature osteoblasts, move into developing and fractured bones along with invading blood vessels.

6. CXCR2 and CXCR4 antagonistically regulate neutrophil trafficking from murine bone marrow.

7. Increased skeletal VEGF enhances beta-catenin activity and results in excessively ossified bones.

8. Endogenous glucocorticoids decrease skeletal angiogenesis, vascularity, hydration, and strength in aged mice.

9. Continuous elevation of PTH increases the number of osteoblasts via both osteoclast-dependent and -independent mechanisms.

10. Ephrin-B2 regulates endothelial cell morphology and motility independently of Eph-receptor binding.

Review 1. Adynamic bone disease is a predominant bone pattern in early stages of chronic kidney disease.

2. Calcium intake is not associated with increased coronary artery calcification: the Framingham Study.

3. FGF2 crosstalk with Wnt signaling in mediating the anabolic action of PTH on bone formation.

4. Case report: extreme coronary calcifications and hypomagnesemia in a patient with a 17q12 deletion involving HNF1B.