Literature DB >> 21911584

Long-term follow-up of the human phenotype in three siblings with cone dystrophy associated with a homozygous p.G461R mutation of KCNV2.

Christoph Friedburg1, Bernd Wissinger, Maria Schambeck, Michael Bonin, Susanne Kohl, Birgit Lorenz.   

Abstract

PURPOSE: To provide an up to 14-year overview of the early ocular phenotype in siblings with a homozygous p.G461R mutation in the KCNV2 gene.
METHODS: Two brothers and their sister were followed-up clinically from ages 5 years, 4 years, and 2 months, respectively, including complete ophthalmological examinations. Goldmann visual fields, two-color-threshold (2CT) perimetry, color vision testing, optical coherence tomography (OCT), fundus autofluorescence (FAF), and Ganzfeld electroretinograms (ERGs) were performed according to age-related capabilities. Genetic analyses included whole genome linkage analysis, homozygosity mapping, and candidate gene sequencing.
RESULTS: All three siblings were homozygous for the p.G461R mutation. At 5 months, the younger brother had no nystagmus and Teller-acuity of 3.2 cyc/deg. At older age, all three presented nystagmus, increased light sensitivity, reduced color discrimination, and relative central scotomas. Visual acuities ranged from 20/200 to 20/70. The macula developed minor irregularities of the RPE, thinning in optical coherence tomography, and a ring of increased FAF. Scotopic (rod) sensitivity was reduced by 2 log and photopic sensitivity by 1 log in two-color-threshold perimetry. ERG responses were markedly delayed. Photopic amplitudes were severely reduced. Scotopic b-waves rose steeply with flash intensity, but for the standard flash supernormal amplitudes were only reached in the girl.
CONCLUSIONS: FAF was similar to that in cone-rod dystrophy. Although cone dysfunction was accompanied by rod dysfunction, and scotopic ERGs in patient 2 deteriorated, no patient demonstrated any unequivocal sign of rod degeneration. Grossly delayed b-waves with a steep response-versus-intensity relationship rather than supernormal amplitudes should remind clinicians of this specific condition.

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Year:  2011        PMID: 21911584     DOI: 10.1167/iovs.11-8187

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  9 in total

1.  Two-color pupillometry in KCNV2 retinopathy.

Authors:  Frederick T Collison; Jason C Park; Gerald A Fishman; Edwin M Stone; J Jason McAnany
Journal:  Doc Ophthalmol       Date:  2019-03-29       Impact factor: 2.379

2.  Phenotypic characteristics including in vivo cone photoreceptor mosaic in KCNV2-related "cone dystrophy with supernormal rod electroretinogram".

Authors:  Ajoy Vincent; Tom Wright; Yaiza Garcia-Sanchez; Marsha Kisilak; Melanie Campbell; Carol Westall; Elise Héon
Journal:  Invest Ophthalmol Vis Sci       Date:  2013-01-30       Impact factor: 4.799

3.  KCNV2-Associated Retinopathy: Genetics, Electrophysiology, and Clinical Course-KCNV2 Study Group Report 1.

Authors:  Michalis Georgiou; Anthony G Robson; Kaoru Fujinami; Shaun M Leo; Ajoy Vincent; Fadi Nasser; Thales Antônio Cabral De Guimarães; Samer Khateb; Nikolas Pontikos; Yu Fujinami-Yokokawa; Xiao Liu; Kazushige Tsunoda; Takaaki Hayashi; Mauricio E Vargas; Alberta A H J Thiadens; Emanuel R de Carvalho; Xuan-Thanh-An Nguyen; Gavin Arno; Omar A Mahroo; Maria Inmaculada Martin-Merida; Belen Jimenez-Rolando; Gema Gordo; Ester Carreño; Ayuso Carmen; Dror Sharon; Susanne Kohl; Rachel M Huckfeldt; Bernd Wissinger; Camiel J F Boon; Eyal Banin; Mark E Pennesi; Arif O Khan; Andrew R Webster; Eberhart Zrenner; Elise Héon; Michel Michaelides
Journal:  Am J Ophthalmol       Date:  2020-12-11       Impact factor: 5.258

4.  Molecular characteristics of four Japanese cases with KCNV2 retinopathy: report of novel disease-causing variants.

Authors:  Kaoru Fujinami; Kazushige Tsunoda; Natsuko Nakamura; Yu Kato; Toru Noda; Kei Shinoda; Kaoru Tomita; Tetsuhisa Hatase; Tomoaki Usui; Masakazu Akahori; Takeshi Itabashi; Takeshi Iwata; Yoko Ozawa; Kazuo Tsubota; Yozo Miyake
Journal:  Mol Vis       Date:  2013-07-20       Impact factor: 2.367

Review 5.  Kv5, Kv6, Kv8, and Kv9 subunits: No simple silent bystanders.

Authors:  Elke Bocksteins
Journal:  J Gen Physiol       Date:  2016-01-11       Impact factor: 4.086

Review 6.  KCNV2 retinopathy: clinical features, molecular genetics and directions for future therapy.

Authors:  Thales A C De Guimaraes; Michalis Georgiou; Anthony G Robson; Michel Michaelides
Journal:  Ophthalmic Genet       Date:  2020-05-22       Impact factor: 1.803

7.  Rod and cone function in patients with KCNV2 retinopathy.

Authors:  Ditta Zobor; Susanne Kohl; Bernd Wissinger; Eberhart Zrenner; Herbert Jägle
Journal:  PLoS One       Date:  2012-10-15       Impact factor: 3.240

8.  Unexpected Genetic Cause in Two Female Siblings with High Myopia and Reduced Visual Acuity.

Authors:  M N Preising; C Friedburg; W Bowl; B Lorenz
Journal:  Biomed Res Int       Date:  2018-05-23       Impact factor: 3.411

Review 9.  Sensing through Non-Sensing Ocular Ion Channels.

Authors:  Meha Kabra; Bikash Ranjan Pattnaik
Journal:  Int J Mol Sci       Date:  2020-09-21       Impact factor: 6.208

  9 in total

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