Literature DB >> 21901241

Differential role of monocyte subsets in atherosclerosis.

M Hristov1, C Weber.   

Abstract

Endothelial dysfunction and inflammation of the arterial wall continuously drive the development of atherosclerosis. Details regarding the sequential involvement of different monocyte subsets in the pathology of this disease have recently emerged. This review concentrates on major monocyte subpopulations in mouse and men and specifically addresses their phenotype, function and recruitment during primary atherosclerosis as well as their contribution to angiogenesis and tissue regeneration secondary to plaque rupture.

Entities:  

Mesh:

Substances:

Year:  2011        PMID: 21901241     DOI: 10.1160/TH11-07-0500

Source DB:  PubMed          Journal:  Thromb Haemost        ISSN: 0340-6245            Impact factor:   5.249


  39 in total

1.  Changes of monocyte subsets in patients with acute coronary syndrome and correlation with myocardial injury markers.

Authors:  Li Zhu; Yigang Yin; Ruifang Zhou; Jie Lin; Jianming Li; Jun Ye
Journal:  Int J Clin Exp Pathol       Date:  2015-06-01

2.  P-selectin-mediated monocyte-cerebral endothelium adhesive interactions link peripheral organ inflammation to sickness behaviors.

Authors:  Charlotte D'Mello; Kiarash Riazi; Tai Le; Katarzyna M Stevens; Arthur Wang; Derek M McKay; Quentin J Pittman; Mark G Swain
Journal:  J Neurosci       Date:  2013-09-11       Impact factor: 6.167

3.  Severe Aortic Valve Stenosis in Adults is Associated with Increased Levels of Circulating Intermediate Monocytes.

Authors:  Bernd Hewing; Sebastian Chi-Diep Au; Antje Ludwig; Rena Ellerbroek; Phillip van Dijck; Lisa Hartmann; Herko Grubitzsch; Carolin Giannini; Michael Laule; Verena Stangl; Gert Baumann; Karl Stangl
Journal:  J Cardiovasc Transl Res       Date:  2017-01-17       Impact factor: 4.132

4.  DNA methylation profiling reveals differences in the 3 human monocyte subsets and identifies uremia to induce DNA methylation changes during differentiation.

Authors:  Adam M Zawada; Jenny S Schneider; Anne I Michel; Kyrill S Rogacev; Björn Hummel; Nicolas Krezdorn; Soeren Müller; Björn Rotter; Peter Winter; Rima Obeid; Jürgen Geisel; Danilo Fliser; Gunnar H Heine
Journal:  Epigenetics       Date:  2016-03-28       Impact factor: 4.528

5.  Correlation between high density lipoprotein and monocyte subpopulations among stable coronary atherosclerotic heart disease patients.

Authors:  Rong-Hai Yang; Ying-Feng Liu; Xue-Jun Wang; Jian-Guang Liang; Jia-Chao Liu
Journal:  Int J Clin Exp Med       Date:  2015-09-15

Review 6.  Preclinical mouse models and methods for the discovery of the causes and treatments of atherosclerosis.

Authors:  Bernd Hewing; Edward A Fisher
Journal:  Expert Opin Drug Discov       Date:  2012-02-10       Impact factor: 6.098

7.  Cyclooxygenase 2, toll-like receptor 4 and interleukin 1β mRNA expression in atherosclerotic plaques of type 2 diabetic patients.

Authors:  Alessandro Baldan; Silvia Ferronato; Silvia Olivato; Giovanni Malerba; Alberto Scuro; Gian Franco Veraldi; Matteo Gelati; Sergio Ferrari; Sara Mariotto; Pier Franco Pignatti; Sara Mazzucco; Macarena Gomez-Lira
Journal:  Inflamm Res       Date:  2014-08-06       Impact factor: 4.575

Review 8.  Neuroimmunology of the atherosclerotic plaque: a morphological approach.

Authors:  Rita Businaro
Journal:  J Neuroimmune Pharmacol       Date:  2012-11-14       Impact factor: 4.147

9.  Differential TNF production by monocyte subsets under physical stress: blunted mobilization of proinflammatory monocytes in prehypertensive individuals.

Authors:  Stoyan Dimitrov; Farah Shaikh; Christopher Pruitt; Michael Green; Kathleen Wilson; Nuzhat Beg; Suzi Hong
Journal:  Brain Behav Immun       Date:  2012-10-06       Impact factor: 7.217

Review 10.  Tetrahydrobiopterin in cardiovascular health and disease.

Authors:  Jennifer K Bendall; Gillian Douglas; Eileen McNeill; Keith M Channon; Mark J Crabtree
Journal:  Antioxid Redox Signal       Date:  2014-03-14       Impact factor: 8.401

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.