Literature DB >> 21858745

H1N1 influenza: critical care aspects.

Glen P Westall1, Miranda Paraskeva.   

Abstract

During the Northern Hemisphere spring of 2009, a novel H1N1 influenza A virus emerged in Mexico, causing widespread human infection and acute critical respiratory illness. The 2009 H1N1 virus spread initially to the United States and Canada, with subsequent rapid global dissemination, leading the World Health Organization (WHO) to declare "a public health emergency of international concern" in April 2009, and upgrading the viral threat to pandemic status in June 2009. Despite initial fears, the severity of the 2009 H1N1 pandemic overall did not differ significantly from that of seasonal influenza. However, the demographics of those at risk of severe illness did differ (affecting children and young adults, rather than the very young and the very old). The 2009 H1N1 pandemic led to rapid implementation of health care initiatives, including the provision of critical care services, to limit the effect of the influenza outbreak on the community. This review focuses on the critical care response to the H1N1 pandemic and examines whether the implementation of critical care services as planned a priori matched the reality of the clinical workload and the patient burden that transpired during the 2009 H1N1 influenza pandemic. © Thieme Medical Publishers.

Entities:  

Mesh:

Year:  2011        PMID: 21858745     DOI: 10.1055/s-0031-1283280

Source DB:  PubMed          Journal:  Semin Respir Crit Care Med        ISSN: 1069-3424            Impact factor:   3.119


  7 in total

1.  Postinfection A77-1726 treatment improves cardiopulmonary function in H1N1 influenza-infected mice.

Authors:  Famke Aeffner; Anna Bratasz; Emilio Flaño; Kimerly A Powell; Ian C Davis
Journal:  Am J Respir Cell Mol Biol       Date:  2012-06-07       Impact factor: 6.914

2.  Activation of A1-adenosine receptors promotes leukocyte recruitment to the lung and attenuates acute lung injury in mice infected with influenza A/WSN/33 (H1N1) virus.

Authors:  Famke Aeffner; Parker S Woods; Ian C Davis
Journal:  J Virol       Date:  2014-06-25       Impact factor: 5.103

3.  Increased expression of microRNA-155-5p by alveolar type II cells contributes to development of lethal ARDS in H1N1 influenza A virus-infected mice.

Authors:  Parker S Woods; Lauren M Doolittle; Lucia E Rosas; S Patrick Nana-Sinkam; Esmerina Tili; Ian C Davis
Journal:  Virology       Date:  2020-03-23       Impact factor: 3.513

4.  TGF-β-induced IL-6 prevents development of acute lung injury in influenza A virus-infected F508del CFTR-heterozygous mice.

Authors:  Parker S Woods; Mia F Tazi; Nicholas M Chesarino; Amal O Amer; Ian C Davis
Journal:  Am J Physiol Lung Cell Mol Physiol       Date:  2015-04-03       Impact factor: 5.464

Review 5.  Introduction and executive summary: care of the critically ill and injured during pandemics and disasters: CHEST consensus statement.

Authors:  Michael D Christian; Asha V Devereaux; Jeffrey R Dichter; Lewis Rubinson; Niranjan Kissoon
Journal:  Chest       Date:  2014-10       Impact factor: 9.410

6.  Can breathing circuit filters help prevent the spread of influenza A (H1N1) virus from intubated patients?

Authors:  Jan F Heuer; Thomas A Crozier; Glenn Howard; Michael Quintel
Journal:  GMS Hyg Infect Control       Date:  2013-04-29

7.  Sentinel surveillance of influenza in Burkina Faso: identification of circulating strains during 2010-2012.

Authors:  Zékiba Tarnagda; Issaka Yougbaré; Abdoul K Ilboudo; Thérèse Kagoné; Armel M Sanou; Assana Cissé; Isaïe Médah; Denis Yelbéogo; Ndahwouh Talla Nzussouo
Journal:  Influenza Other Respir Viruses       Date:  2014-07-29       Impact factor: 4.380

  7 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.