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21 in total

1. Persisting risk of nickel related lung cancer and nasal cancer among Clydach refiners.

Authors: T K Grimsrud; J Peto
Journal: Occup Environ Med Date: 2006-05 Impact factor: 4.402

2. Soluble and insoluble nickel compounds exert a differential inhibitory effect on cell growth through IKKalpha-dependent cyclin D1 down-regulation.

Authors: Weiming Ouyang; Dongyun Zhang; Jingxia Li; Udit N Verma; Max Costa; Chuanshu Huang
Journal: J Cell Physiol Date: 2009-01 Impact factor: 6.384

3. Mortality experience among employees at a hydrometallurgical nickel refinery and fertiliser complex in Fort Saskatchewan, Alberta (1954-95).

Authors: R Egedahl; M Carpenter; D Lundell
Journal: Occup Environ Med Date: 2001-11 Impact factor: 4.402

Review 4. The control of histone methylation and gene expression by oxidative stress, hypoxia, and metals.

Authors: Yana Chervona; Max Costa
Journal: Free Radic Biol Med Date: 2012-07-25 Impact factor: 7.376

5. Molecular biology of nickel carcinogenesis: identification of differentially expressed genes in morphologically transformed C3H10T1/2 Cl 8 mouse embryo fibroblast cell lines induced by specific insoluble nickel compounds.

Authors: Rini Verma; Jamuna Ramnath; Farrah Clemens; Lisa C Kaspin; Joseph R Landolph
Journal: Mol Cell Biochem Date: 2004-01 Impact factor: 3.396

10. New views on the hypothesis of respiratory cancer risk from soluble nickel exposure; and reconsideration of this risk's historical sources in nickel refineries.

Authors: Philip G Thornhill; Bruce R Conard; James G Heller
Journal: J Occup Med Toxicol Date: 2009-08-23 Impact factor: 2.646

Report of the International Committee on Nickel Carcinogenesis in Man.

1. Persisting risk of nickel related lung cancer and nasal cancer among Clydach refiners.

2. Soluble and insoluble nickel compounds exert a differential inhibitory effect on cell growth through IKKalpha-dependent cyclin D1 down-regulation.

3. Mortality experience among employees at a hydrometallurgical nickel refinery and fertiliser complex in Fort Saskatchewan, Alberta (1954-95).

Review 4. The control of histone methylation and gene expression by oxidative stress, hypoxia, and metals.

5. Molecular biology of nickel carcinogenesis: identification of differentially expressed genes in morphologically transformed C3H10T1/2 Cl 8 mouse embryo fibroblast cell lines induced by specific insoluble nickel compounds.

6. Evidence of carcinogenicity in humans of water-soluble nickel salts.

7. Contamination of the Conchos River in Mexico: does it pose a health risk to local residents?

8. In vitro and in vivo uptake of nickel sulfides by rat lymphocytes.

9. Nickel compounds induce histone ubiquitination by inhibiting histone deubiquitinating enzyme activity.

10. New views on the hypothesis of respiratory cancer risk from soluble nickel exposure; and reconsideration of this risk's historical sources in nickel refineries.