Literature DB >> 21778257

Portopulmonary hypertension: a report from the US-based REVEAL Registry.

Michael J Krowka1, Dave P Miller2, Robyn J Barst3, Darren Taichman4, Raed A Dweik5, David B Badesch6, Michael D McGoon7.   

Abstract

BACKGROUND: We evaluated survival and hospitalization rates in patients with group 1 portopulmonary hypertension (PoPH) in the Registry to Evaluate Early and Long-term Pulmonary Arterial Hypertension Disease Management (REVEAL Registry).
METHODS: The REVEAL Registry is a multicenter, observational, US-based study evaluating demographics and management of patients with pulmonary arterial hypertension (PAH). Outcomes were examined using Kaplan-Meier time-to-event estimates and compared with patients with idiopathic PAH (IPAH) or familial PAH (FPAH).
RESULTS: One hundred seventy-four patients with PoPH were enrolled in the REVEAL Registry (IPAH/FPAH; n = 1,478) from March 2006 to December 2009. Mean age was 53 ± 10 years, 52% were female, 32% were newly diagnosed, and 6% were New York Heart Association/World Health Organization functional class IV. Outcome parameters were worse for PoPH vs IPAH/FPAH, respectively: 2-year survival from enrollment (67% vs 85%, P < .001), 5-year survival from time of diagnosis (40% vs 64%, P < .001), and 2-year freedom from all-cause hospitalization (49% vs 59%, P = .019). However, despite worse outcomes, hemodynamic parameters at diagnosis were better for PoPH vs IPAH/FPAH, respectively: mean pulmonary artery pressure (49 mm Hg vs 53 mm Hg, P < .001), mean right atrial pressure (9 mm Hg vs 10 mm Hg, P = .005), pulmonary vascular resistance (8 Wood units vs 12 Wood units, P < .001), and cardiac output (5 L/min vs 4 L/min, P < .001). Compared with patients with IPAH/FPAH, patients with PoPH were less likely to be on a PAH-specific therapy at enrollment (P < .001), suggesting potential delays in therapy for patients with PoPH.
CONCLUSIONS: Patients with PoPH had significantly poorer survival and all-cause hospitalization rates compared with patients with IPAH/FPAH, despite having better hemodynamics at diagnosis. Further studies should investigate such outcomes and differences in treatment patterns. TRIAL REGISTRY: ClinicalTrials.gov; No.: NCT00370214; URL: www.clinicaltrials.gov.

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Year:  2011        PMID: 21778257     DOI: 10.1378/chest.11-0160

Source DB:  PubMed          Journal:  Chest        ISSN: 0012-3692            Impact factor:   9.410


  54 in total

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3.  Acute right ventricular failure after orthotopic liver transplantation.

Authors:  Rohan M Goswami; Rami N Khouzam
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4.  Effectiveness of phosphodiesterase-5 inhibitor therapy for portopulmonary hypertension.

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5.  Hemodynamic effects of ambrisentan-tadalafil combination therapy on progressive portopulmonary hypertension.

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Review 6.  Sex, Gender, and Sex Hormones in Pulmonary Hypertension and Right Ventricular Failure.

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7.  Portopulmonary hypertension: improved detection using CT and echocardiography in combination.

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Review 8.  Portopulmonary hypertension and hepatopulmonary syndrome.

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9.  An official American Thoracic Society Statement: pulmonary hypertension phenotypes.

Authors:  Raed A Dweik; Sharon Rounds; Serpil C Erzurum; Stephen Archer; Karen Fagan; Paul M Hassoun; Nicholas S Hill; Marc Humbert; Steven M Kawut; Michael Krowka; Evangelos Michelakis; Nicholas W Morrell; Kurt Stenmark; Rubin M Tuder; John Newman
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Review 10.  Pulmonary vascular complications of liver disease.

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