Literature DB >> 21760507

Current management of lymphangioleiomyomatosis.

Camille Taillé1, Raphaël Borie, Bruno Crestani.   

Abstract

PURPOSE OF REVIEW: Lymphangioleiomyomatosis (LAM) is a rare but devastating disease, leading to chronic respiratory failure. Considerable progress for comprehension of the disease has been made when mutations of the tuberous sclerosis genes TSC1 and TSC2, were discovered in LAM cells. Therapeutic consequences of these studies are important, leading to clinical trials with sirolimus for LAM. RECENT
FINDINGS: In two studies, angiomyolipoma size decreased by 26-50% after 12 months of sirolimus treatment. In a recent 12 months controlled trial involving 89 patients with pulmonary LAM, sirolimus stopped lung function decline and improved quality of life and performance score. The protective effect of sirolimus was lost after treatment discontinuation, with a parallel lung function decline in both groups, similar to the increase in angiomyolipoma size. Sirolimus is associated with an excess of adverse events.
SUMMARY: Sirolimus represents an important drug for LAM that should be proposed to patients with a rapid alteration of lung function or with a significant clinical impairment, after individual evaluation of the risk/benefit ratio. Sirolimus seems to have a sharper effect on the reduction of abdominal masses than on lung cysts. Tolerance and safety concerns are serious limits to the long-term treatment of patients with sirolimus.

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Year:  2011        PMID: 21760507     DOI: 10.1097/MCP.0b013e328349ac8c

Source DB:  PubMed          Journal:  Curr Opin Pulm Med        ISSN: 1070-5287            Impact factor:   3.155


  7 in total

1.  Lymphangioleiomyomatosis: current and future.

Authors:  Maria Mavroudi; Paul Zarogoulidis; Nikolaos Katsikogiannis; Kosmas Tsakiridis; Haidong Huang; Antonios Sakkas; Anastasios Kallianos; Aggeliki Rapti; Eirini Sarika; Ilias Karapantzos; Konstantinos Zarogoulidis
Journal:  J Thorac Dis       Date:  2013-02       Impact factor: 2.895

2.  Restoration of Corticosteroid Sensitivity in Chronic Obstructive Pulmonary Disease by Inhibition of Mammalian Target of Rapamycin.

Authors:  Akihisa Mitani; Kazuhiro Ito; Chaitanya Vuppusetty; Peter J Barnes; Nicolas Mercado
Journal:  Am J Respir Crit Care Med       Date:  2016-01-15       Impact factor: 21.405

3.  Adolescent form of sporadic lymphangioleiomyomatosis (S-LAM).

Authors:  S M Restrepo-Gualteros; C E Rodriguez-Martinez; L E Jaramillo-Barberi; A F Lopez-Cadena; G Nino
Journal:  Allergol Immunopathol (Madr)       Date:  2014-05-01       Impact factor: 1.667

4.  Uterine-specific loss of Tsc2 leads to myometrial tumors in both the uterus and lungs.

Authors:  Hen Prizant; Aritro Sen; Allison Light; Sung-Nam Cho; Francesco J DeMayo; John P Lydon; Stephen R Hammes
Journal:  Mol Endocrinol       Date:  2013-07-02

5.  Integration of mTOR and estrogen-ERK2 signaling in lymphangioleiomyomatosis pathogenesis.

Authors:  Xiaoxiao Gu; Jane J Yu; Didem Ilter; Nickolas Blenis; Elizabeth Petri Henske; John Blenis
Journal:  Proc Natl Acad Sci U S A       Date:  2013-08-27       Impact factor: 11.205

6.  Understanding the priorities for women diagnosed with lymphangioleiomyomatosis: a patient perspective.

Authors:  Iris Bassi; Gill Hollis; Vincent Cottin; Sergio Harari; Elma Zwanenburg; Marcel Veltkamp; Alvaro Casanova; Monica Fletcher; Sarah Masefield; Pippa Powell; Jeanette Boyd
Journal:  ERJ Open Res       Date:  2016-04-21

7.  Mutation spectrums of TSC1 and TSC2 in Chinese women with lymphangioleiomyomatosis (LAM).

Authors:  Jie Liu; Weiwei Zhao; Xiaohua Ou; Zhen Zhao; Changming Hu; Mingming Sun; Feifei Liu; Junhao Deng; Weili Gu; Jiaying An; Qingling Zhang; Xiaoxian Zhang; Jiaxing Xie; Shiyue Li; Rongchang Chen; Shihui Yu; Nanshan Zhong
Journal:  PLoS One       Date:  2019-12-19       Impact factor: 3.240

  7 in total

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