Literature DB >> 21741045

Adiponectin: an independent risk factor for coronary heart disease in men in the Framingham offspring Study.

Masumi Ai1, Seiko Otokozawa, Bela F Asztalos, Charles C White, L Adrienne Cupples, Katsuyuki Nakajima, Stefania Lamon-Fava, Peter W Wilson, Yuji Matsuzawa, Ernst J Schaefer.   

Abstract

OBJECTIVE: Our aim was to determine whether plasma adiponectin levels were an independent predictor of coronary heart disease (CHD) risk. METHODS AND
RESULTS: Plasma adiponectin levels were measured in 3188 male and female participants from cycle 6 of the Framingham offspring Study (mean age: 57 years in both men and women; BMI: 28.5 kg/m(2) in men and 27.3 kg/m(2) in women), using a novel fully automated assay. Plasma adiponectin levels (median [25th percentile, 75th percentile]) were significantly higher in female than in male CHD-free subjects (14.8 [10.7,20.5] μg/ml versus 9.0 [7.0,12.2] μg/ml, p<0.001). Participants were followed for a mean of 7.5 years. After adjustment for age, BMI, smoking status, systolic blood pressure, treatment for hypertension, diabetes, use of cholesterol-lowering medication, total cholesterol level, high-density lipoprotein cholesterol level, and C-reactive protein levels, a higher plasma adiponectin level was a significant predictor of lower risk of future CHD events (n=117) in men (HR 0.49, p<0.0022). A similar trend was observed in women, but was no longer significant after multivariate adjustments.
CONCLUSIONS: Our data indicate that plasma adiponectin levels are an independent predictor of CHD in Caucasian men initially free of CHD.
Copyright © 2011. Published by Elsevier Ireland Ltd.

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Year:  2011        PMID: 21741045      PMCID: PMC3662557          DOI: 10.1016/j.atherosclerosis.2011.05.035

Source DB:  PubMed          Journal:  Atherosclerosis        ISSN: 0021-9150            Impact factor:   5.162


  27 in total

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9.  Hypoadiponectinemia as an independent predictor for the progression of carotid atherosclerosis: a 5-year prospective study.

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10.  Associations between two single nucleotide polymorphisms of the adiponectin gene, its circulating concentrations and cardiometabolic risk factors in prepubertal children with and without abdominal obesity.

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