Literature DB >> 21719078

The effects of an anti-IL-13 mAb on cytokine levels and nasal symptoms following nasal allergen challenge.

Grant C Nicholson1, Harsha H Kariyawasam, Andrew J Tan, Jens M Hohlfeld, Deborah Quinn, Christoph Walker, David Rodman, John Westwick, Stipo Jurcevic, Onn Min Kon, Peter J Barnes, Norbert Krug, Trevor T Hansel.   

Abstract

BACKGROUND: IL-13 is a key T(H)2 cytokine that is implicated in allergic responses.
OBJECTIVE: We evaluated the effects of an anti-IL-13-blocking antibody compared with placebo on repeated nasal allergen challenge responses in hay fever patients out of season.
METHODS: We performed a parallel group double-blind study of anti-IL-13 (single dose, 6 mg/kg intravenously, n = 16) and placebo (n = 15), with an additional open label group given a topical nasal corticosteroid (n = 5). Subjects received intranasal timothy grass pollen (Phleum pratense P5 allergen), and serial samples of nasal mucosal lining fluid were taken by using synthetic absorptive matrix and by nasal lavage.
RESULTS: Administration of anti-IL-13 on day 1 resulted in a significant decrease in IL-13 levels in synthetic absorptive matrix eluates compared with placebo (area under the curve 0-8 hours, change from baseline) during the late phase after nasal allergen challenge on day 5 (P < .05) and day 7 (P < .01). There were no apparent effects of anti-IL-13 treatment on nasal lavage eosinophil numbers or total nasal symptom scores versus placebo. However, in a subgroup with high late-phase IL-13 levels at screening, there was a trend for a decrease in total nasal symptom scores after nasal allergen challenge on day 5, when compared with subjects with low IL-13 levels (P < .10). Nasal fluticasone caused suppression of IL-13 (P < .05 on day 5) as well as IL-5 (P < .01 on day 5) levels in the late phase compared with placebo.
CONCLUSIONS: Anti-IL-13 had specific pharmacodynamic action in this nasal allergen challenge model, causing profound inhibition of nasal lining fluid IL-13 responses. In addition, there was a possible effect of anti-IL-13 treatment on total nasal symptom scores in a subgroup with high late-phase nasal IL-13 levels at screening.
Copyright © 2011 American Academy of Allergy, Asthma & Immunology. Published by Mosby, Inc. All rights reserved.

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Year:  2011        PMID: 21719078     DOI: 10.1016/j.jaci.2011.05.013

Source DB:  PubMed          Journal:  J Allergy Clin Immunol        ISSN: 0091-6749            Impact factor:   10.793


  22 in total

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Review 4.  Targeting eosinophils in allergy, inflammation and beyond.

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5.  IL-17A induces signal transducers and activators of transcription-6-independent airway mucous cell metaplasia.

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Review 6.  Re-educating immunity in respiratory allergies: the potential for hematopoietic stem cell-mediated gene therapy.

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Review 8.  Th17-mediated inflammation in asthma.

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Review 9.  Therapies for allergic inflammation: refining strategies to induce tolerance.

Authors:  Cezmi A Akdis
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10.  IL-33-dependent type 2 inflammation during rhinovirus-induced asthma exacerbations in vivo.

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Journal:  Am J Respir Crit Care Med       Date:  2014-12-15       Impact factor: 21.405

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