Literature DB >> 21635296

Combining versus analyzing multiple causes: how domain assumptions and task context affect integration rules.

Michael R Waldmann1.   

Abstract

In everyday life, people typically observe fragments of causal networks. From this knowledge, people infer how novel combinations of causes they may never have observed together might behave. I report on 4 experiments that address the question of how people intuitively integrate multiple causes to predict a continuously varying effect. Most theories of causal induction in psychology and statistics assume a bias toward linearity and additivity. In contrast, these experiments show that people are sensitive to cues biasing various integration rules. Causes that refer to intensive quantities (e.g., taste) or to preferences (e.g., liking) bias people toward averaging the causal influences, whereas extensive quantities (e.g., strength of a drug) lead to a tendency to add. However, the knowledge underlying these processes is fallible and unstable. Therefore, people are easily influenced by additional task-related context factors. These additional factors include the way data are presented, the difficulty of the inference task, and transfer from previous tasks. The results of the experiments provide evidence for causal model and related theories, which postulate that domain-general representations of causal knowledge are influenced by abstract domain knowledge, data-driven task factors, and processing difficulty. 2007 Cognitive Science Society, Inc.

Entities:  

Year:  2007        PMID: 21635296     DOI: 10.1080/15326900701221231

Source DB:  PubMed          Journal:  Cogn Sci        ISSN: 0364-0213


  13 in total

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7.  Effect of grouping of evidence types on learning about interactions between observed and unobserved causes.

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8.  Testing the deductive inferential account of blocking in causal learning.

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9.  Causal explanation in the face of contradiction.

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10.  Blocking in human causal learning is affected by outcome assumptions manipulated through causal structure.

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