Literature DB >> 21623190

The aetiology of symptomatic gallstones quantification of the effects of obesity, alcohol and serum lipids on risk. Epidemiological and biomarker data from a UK prospective cohort study (EPIC-Norfolk).

Paul J R Banim1, Robert N Luben, Heerajnarain Bulluck, Stephen J Sharp, Nicholas J Wareham, Kay-Tee Khaw, Andrew R Hart.   

Abstract

OBJECTIVE: The development of gallstones is influenced by obesity and alcohol. This study aimed to precisely quantify these risks and investigate whether the aetiological mechanism may involve serum lipids, for the first time using a European prospective cohort study.
METHODS: The European Prospective Investigation into Cancer-Norfolk, recruited 25 639 men and women, aged 40 to 74 years, between 1993 and 1997. At enrolment weight, height and alcohol intake were recorded and nonfasting blood samples taken to measure serum triglycerides, cholesterol, high-density lipoproteins and low-density lipoproteins. The cohort was monitored for 14 years for symptomatic gallstones. Cox regression estimated sex-specific hazard ratios (HRs) for symptomatic gallstones adjusted for covariates.
RESULTS: Symptomatic gallstones developed in 296 people (67.9% women). For each additional unit of BMI, the HR in men was 1.08 [95% confidence interval (CI)=1.02-1.14]; in women the HR was 1.08 (95% CI=1.06-1.11). Every unit of alcohol consumed per week decreased risk in men by 3% (HR=0.97, 95% CI=0.95-0.99) with no effect in women. Serum triglycerides increased risk in men (highest vs. lowest quarter HR=2.02, 95% CI=1.03-3.98) and women (HR=2.43, 95% CI=1.52-3.90). Increased high-density lipoprotein was associated with a decreased risk in men (highest vs. lowest quarter HR=0.22, 95% CI=0.09-0.52) and women (HR=0.55, 95% CI=0.36-0.85). No effects were found for serum cholesterol and low-density lipoprotein.
CONCLUSION: Obesity and alcohol influence gallstone formation, possibly in part through their effects on serum lipids. Reducing obesity may prevent gallstones in the population, as 38% of incident cases of gallstones were associated with a BMI of more than 25.

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Year:  2011        PMID: 21623190     DOI: 10.1097/MEG.0b013e3283477cc9

Source DB:  PubMed          Journal:  Eur J Gastroenterol Hepatol        ISSN: 0954-691X            Impact factor:   2.566


  14 in total

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2.  ABCG1 regulates mouse adipose tissue macrophage cholesterol levels and ratio of M1 to M2 cells in obesity and caloric restriction.

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Review 4.  The Role of Diet in the Pathogenesis of Cholesterol Gallstones.

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Journal:  Curr Med Chem       Date:  2019       Impact factor: 4.530

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Review 7.  Involvement of inflammatory factors in pancreatic carcinogenesis and preventive effects of anti-inflammatory agents.

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Review 8.  Tobacco smoking and the risk of gallbladder disease.

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Review 10.  Preventing a Mass Disease: The Case of Gallstones Disease: Role and Competence for Family Physicians.

Authors:  Piero Portincasa; Agostino Di Ciaula; Ignazio Grattagliano
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