Literature DB >> 21565796

Combination of niacin and fenofibrate with lifestyle changes improves dyslipidemia and hypoadiponectinemia in HIV patients on antiretroviral therapy: results of "heart positive," a randomized, controlled trial.

Ashok Balasubramanyam1, Ivonne Coraza, E O'Brian Smith, Lynne W Scott, Payal Patel, Dinakar Iyer, Addison A Taylor, Thomas P Giordano, Rajagopal V Sekhar, Pamela Clark, Edith Cuevas-Sanchez, Swarna Kamble, Christie M Ballantyne, Henry J Pownall.   

Abstract

CONTEXT: HIV patients on antiretroviral therapy (ART) have a unique dyslipidemia [elevated triglycerides and non-high-density lipoprotein-cholesterol (HDL-C), low HDL-C] with insulin resistance (characterized by hypoadiponectinemia).
OBJECTIVE: The aim was to test a targeted, comprehensive, additive approach to treating the dyslipidemia. DESIGN AND
SETTING: We conducted a randomized, double-blind, placebo-controlled, 24-wk trial of lifestyle modification, fenofibrate, and niacin in multiethnic HIV clinics at an academic center. PARTICIPANTS: Hypertriglyceridemic adult patients were stratified on three combinations of ART classes. Subjects retained at the first measurement (2 wk) after entry were included in the analysis (n = 191).
INTERVENTIONS: Subjects were randomized into five treatment groups: usual care (group 1); low-saturated-fat diet and exercise (D/E; group 2); D/E + fenofibrate (group 3); D/E + niacin (group 4); or D/E + fenofibrate + niacin (group 5). MAIN OUTCOME MEASURES: We measured changes in fasting triglycerides, HDL-C, and non-HDL-C (primary), and in insulin sensitivity, glycemia, adiponectin, C-reactive protein, energy expenditure, and body composition (secondary). Data were analyzed as a factorial set of treatment combinations using a mixed repeated measures model, last observation carried forward, and complete case approaches (groups 2-5), and as an unstructured set of treatments (groups 1-5).
RESULTS: Fenofibrate improved triglycerides (P = 0.002), total cholesterol (P = 0.02), and non-HDL-C (P = 0.003), whereas niacin improved HDL-C (P = 0.03), and both drugs decreased the total cholesterol-to-HDL-C ratio (P = 0.005-0.01). The combination of D/E, fenofibrate, and niacin provided maximal benefit, markedly reducing triglycerides (-52% compared to usual care; P = 0.003), increasing HDL-C (+12%; P < 0.001), and decreasing non-HDL-C (-18.5%; P = 0.003) and total cholesterol-to-HDL-C ratio (-24.5%; P < 0.001). Niacin doubled adiponectin levels.
CONCLUSIONS: A combination of fenofibrate and niacin with low-saturated-fat D/E is effective and safe in increasing HDL-C, decreasing non-HDL-C and hypertriglyceridemia, and ameliorating hypoadiponectinemia in patients with HIV/ART-associated dyslipidemia.

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Year:  2011        PMID: 21565796      PMCID: PMC3135191          DOI: 10.1210/jc.2010-3067

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  45 in total

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2.  Simvastatin and niacin, antioxidant vitamins, or the combination for the prevention of coronary disease.

Authors:  B G Brown; X Q Zhao; A Chait; L D Fisher; M C Cheung; J S Morse; A A Dowdy; E K Marino; E L Bolson; P Alaupovic; J Frohlich; J J Albers
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Review 3.  The paradox of improved antiretroviral therapy in HIV: potential for nutritional modulation?

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4.  Effects of combination lipid therapy in type 2 diabetes mellitus.

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Journal:  N Engl J Med       Date:  2010-03-14       Impact factor: 91.245

5.  Effects of protease inhibitors on hyperglycemia, hyperlipidemia, and lipodystrophy: a 5-year cohort study.

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6.  Niacin stimulates adiponectin secretion through the GPR109A receptor.

Authors:  Eric P Plaisance; Martina Lukasova; Stefan Offermanns; Youyan Zhang; Guoqing Cao; Robert L Judd
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Journal:  JAMA       Date:  2009-11-11       Impact factor: 56.272

Review 8.  Review of extended-release niacin/laropiprant fixed combination in the treatment of mixed dyslipidemia and primary hypercholesterolemia.

Authors:  Klaus G Parhofer
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9.  Metabolic basis of HIV-lipodystrophy syndrome.

Authors:  Rajagopal V Sekhar; Farook Jahoor; A Clinton White; Henry J Pownall; Fehmida Visnegarwala; Maria C Rodriguez-Barradas; Morali Sharma; Peter J Reeds; Ashok Balasubramanyam
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10.  Risk of myocardial infarction in patients with HIV infection exposed to specific individual antiretroviral drugs from the 3 major drug classes: the data collection on adverse events of anti-HIV drugs (D:A:D) study.

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Journal:  J Infect Dis       Date:  2010-02-01       Impact factor: 5.226

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  25 in total

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Review 2.  Niacin for primary and secondary prevention of cardiovascular events.

Authors:  Stefan Schandelmaier; Matthias Briel; Ramon Saccilotto; Kelechi K Olu; Armon Arpagaus; Lars G Hemkens; Alain J Nordmann
Journal:  Cochrane Database Syst Rev       Date:  2017-06-14

3.  Therapy: HIV-associated dyslipidemia: the heart positive study.

Authors:  Steven Grinspoon; Kathleen Fitch
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4.  Extended-Release Niacin Versus Fenofibrate in HIV-Infected Participants With Low High-Density Lipoprotein Cholesterol: Effects on Endothelial Function, Lipoproteins, and Inflammation.

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Review 5.  Interventions to address chronic disease and HIV: strategies to promote exercise and nutrition among HIV-infected individuals.

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6.  Effects of lifestyle modification and metformin on atherosclerotic indices among HIV-infected patients with the metabolic syndrome.

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7.  Intensive lifestyle modification reduces Lp-PLA2 in dyslipidemic HIV/HAART patients.

Authors:  Joshua S Wooten; Preethi Nambi; Baiba K Gillard; Henry J Pownall; Ivonne Coraza; Lynne W Scott; Vijay Nambi; Christie M Ballantyne; Ashok Balasubramanyam
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Review 8.  Dysregulation of glucose metabolism in HIV patients: epidemiology, mechanisms, and management.

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9.  Altered relationship of plasma triglycerides to HDL cholesterol in patients with HIV/HAART-associated dyslipidemia: further evidence for a unique form of metabolic syndrome in HIV patients.

Authors:  Catherine N Vu; Raul Ruiz-Esponda; Eric Yang; Evelyn Chang; Baiba Gillard; Henry J Pownall; Ron C Hoogeveen; Ivonne Coraza; Ashok Balasubramanyam
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Review 10.  Insulin resistance, lipodystrophy and cardiometabolic syndrome in HIV/AIDS.

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