Literature DB >> 21538529

The LRRK2 R1441C mutation is more frequent than G2019S in Parkinson's disease patients from southern Italy.

Chiara Criscuolo1, Anna De Rosa, Anna Guacci, Erik J Simons, Guido J Breedveld, Silvio Peluso, Giampiero Volpe, Alessandro Filla, Ben A Oostra, Vincenzo Bonifati, Giuseppe De Michele.   

Abstract

BACKGROUND: Mutations in the leucine-rich repeat kinase 2 gene are the most frequent cause of familial and sporadic Parkinson's disease, and G2019S is the most common leucine-rich repeat kinase 2 mutation across several Mediterranean countries.
METHODS: One hundred ninety-two patients with Parkinson's disease from Campania, a region in southern Italy, were screened for R1441C/H/G and G2019S by direct sequencing and SfcI digestion.
RESULTS: Among 192 patients with Parkinson's disease (mean age±SD, 63.9±11.8 years; disease onset, 54.0±12.5 years; family history for Parkinson's disease or tremor, 45%), 8 carried a heterozygous R1441C mutation, whereas only 1 had the G2019S mutation. All R1441C patients originate from the province of Naples and share the same haplotype, suggesting a founder effect.
CONCLUSIONS: G2019S is not ubiquitously the most common leucine-rich repeat kinase 2 mutation; in Campania R1441C is more frequent. Region-specific mutation prevalence data should be taken into account for a sensitive and cost-effective molecular diagnosis and counseling of patients with Parkinson's disease.
Copyright © 2011 Movement Disorder Society.

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Year:  2011        PMID: 21538529     DOI: 10.1002/mds.23735

Source DB:  PubMed          Journal:  Mov Disord        ISSN: 0885-3185            Impact factor:   10.338


  9 in total

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Authors:  Anna De Rosa; Teresa Pellegrino; Sabina Pappatà; Maria Teresa Pellecchia; Silvio Peluso; Francesco Saccà; Paolo Barone; Alberto Cuocolo; Giuseppe De Michele
Journal:  J Nucl Cardiol       Date:  2015-12-01       Impact factor: 5.952

2.  Type II kinase inhibitors show an unexpected inhibition mode against Parkinson's disease-linked LRRK2 mutant G2019S.

Authors:  Min Liu; Samantha A Bender; Gregory D Cuny; Woody Sherman; Marcie Glicksman; Soumya S Ray
Journal:  Biochemistry       Date:  2013-03-01       Impact factor: 3.162

3.  The discovery of LRRK2 p.R1441S, a novel mutation for Parkinson's disease, adds to the complexity of a mutational hotspot.

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Review 4.  Advances in the Genetics of Parkinson's Disease: A Guide for the Clinician.

Authors:  Una-Marie Sheerin; Henry Houlden; Nicholas W Wood
Journal:  Mov Disord Clin Pract       Date:  2014-04-10

Review 5.  Is inhibition of kinase activity the only therapeutic strategy for LRRK2-associated Parkinson's disease?

Authors:  Iakov N Rudenko; Ruth Chia; Mark R Cookson
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Review 6.  Review of the epidemiology and variability of LRRK2 non-p.Gly2019Ser pathogenic mutations in Parkinson's disease.

Authors:  Paweł Turski; Iwona Chaberska; Piotr Szukało; Paulina Pyska; Łukasz Milanowski; Stanisław Szlufik; Monika Figura; Dorota Hoffman-Zacharska; Joanna Siuda; Dariusz Koziorowski
Journal:  Front Neurosci       Date:  2022-09-20       Impact factor: 5.152

7.  Kinase inhibitors arrest neurodegeneration in cell and C. elegans models of LRRK2 toxicity.

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Journal:  Hum Mol Genet       Date:  2012-10-12       Impact factor: 6.150

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Journal:  J Neurol       Date:  2013-02-12       Impact factor: 4.849

9.  LRRK2 BAC transgenic rats develop progressive, L-DOPA-responsive motor impairment, and deficits in dopamine circuit function.

Authors:  Max Sloan; Javier Alegre-Abarrategui; Dawid Potgieter; Anna-Kristin Kaufmann; Richard Exley; Thierry Deltheil; Sarah Threlfell; Natalie Connor-Robson; Katherine Brimblecombe; Rebecca Wallings; Milena Cioroch; David M Bannerman; J Paul Bolam; Peter J Magill; Stephanie J Cragg; Paul D Dodson; Richard Wade-Martins
Journal:  Hum Mol Genet       Date:  2016-01-06       Impact factor: 6.150

  9 in total

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