Literature DB >> 21488058

Extraneuronal toxicity of Alzheimer's β-amyloid peptide: comparative study on vertebrate skeletal muscles.

Marat A Mukhamedyarov1, Alexander Y Teplov, Sergey N Grishin, Alina V Leushina, Andrey L Zefirov, András Palotás.   

Abstract

INTRODUCTION: Alzheimer's β-amyloid peptide (βAP) is known to possess a wide range of toxic effects on neurons in vitro and in vivo; however, there is little information available regarding its impact on other excitable tissues such as skeletal muscles, which, apart from brain cells, are thought to also be targets of βAP.
METHODS: Utilizing the combination of electrophysiology and myography, we investigated whether βAP also impairs the functioning of myocytes in frogs and mice.
RESULTS: Although application of βAP in the range of 10(-6) to 10(-8) M induced depolarization of muscle fibers in both species, it impaired contractility in frogs but not in mice, by reducing endplate potential amplitude and increasing the threshold potential.
CONCLUSIONS: Unchanged contractility in the mouse in the presence of βAP is due to a higher safety factor of neuromuscular transmission in mammals compared with amphibians. Possible clinical implications are discussed.
Copyright © 2011 Wiley Periodicals, Inc.

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Year:  2011        PMID: 21488058     DOI: 10.1002/mus.22000

Source DB:  PubMed          Journal:  Muscle Nerve        ISSN: 0148-639X            Impact factor:   3.217


  4 in total

1.  Amyloid beta peptide 22-35 induces a negative inotropic effect on isolated rat hearts.

Authors:  Neda Yousefirad; Ziya Kaygısız; Yasemin Aydın
Journal:  Int J Physiol Pathophysiol Pharmacol       Date:  2016-12-25

2.  Muscle profile and cognition in patients with Alzheimer's disease dementia.

Authors:  Yeonsil Moon; Ye-Ji Choi; Jin Ok Kim; Seol-Heui Han
Journal:  Neurol Sci       Date:  2018-08-06       Impact factor: 3.307

3.  Mini Nutritional Assessment May Identify a Dual Pattern of Perturbed Plasma Amino Acids in Patients with Alzheimer's Disease: A Window to Metabolic and Physical Rehabilitation?

Authors:  Roberto Aquilani; Alfredo Costa; Roberto Maestri; Matteo Cotta Ramusino; Antonia Pierobon; Maurizia Dossena; Sebastiano Bruno Solerte; Anna Maria Condino; Valeria Torlaschi; Paola Bini; Mirella Boselli; Mauro Ceroni; Daniela Buonocore; Federica Boschi; Mariella Bruni; Manuela Verri
Journal:  Nutrients       Date:  2020-06-21       Impact factor: 5.717

4.  The Transient Receptor Potential Melastatin 2 (TRPM2) Channel Contributes to β-Amyloid Oligomer-Related Neurotoxicity and Memory Impairment.

Authors:  Valeriy G Ostapchenko; Megan Chen; Monica S Guzman; Yu-Feng Xie; Natalie Lavine; Jue Fan; Flavio H Beraldo; Amanda C Martyn; Jillian C Belrose; Yasuo Mori; John F MacDonald; Vania F Prado; Marco A M Prado; Michael F Jackson
Journal:  J Neurosci       Date:  2015-11-11       Impact factor: 6.167

  4 in total

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