Literature DB >> 21445696

Urethral compensatory mechanisms to maintain urinary continence after pudendal nerve injury in female rats.

Akira Furuta1, Yasuyuki Suzuki, Koji Asano, William C de Groat, Shin Egawa, Naoki Yoshimura.   

Abstract

INTRODUCTION AND HYPOTHESIS: This study was conducted to investigate the urethral compensatory mechanisms to maintain urinary continence after pudendal nerve injury.
METHODS: In naive, acute pudendal nerve transection (PNT) and 4 weeks after PNT (PNT-4w) female rats, leak point pressures (LPPs) during bladder compression were measured before and after the application of hexamethonium (C6), propranolol, and N (ω)-nitro-L: -arginine-methyl ester (L: -NAME), or terazosin and atropine. Responses to carbachol and phenylephrine of proximal and middle urethral muscle strips from naive and PNT-4w rats were also examined.
RESULTS: LPPs were significantly decreased in PNT rats but not in PNT-4w rats. LPPs in PNT rats were significantly increased by C6 or L-NAME while LPPs in PNT-4w rats were significantly decreased by C6, or terazosin and atropine. Excitatory responses to carbachol and phenylephrine in the proximal urethral muscle were significantly larger in PNT-4w rats.
CONCLUSIONS: These results suggest that α(1)-adrenoceptor and muscarinic receptor-mediated contractility is upregulated in the proximal urethra 4 weeks after PNT.

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Year:  2011        PMID: 21445696      PMCID: PMC3133823          DOI: 10.1007/s00192-011-1403-6

Source DB:  PubMed          Journal:  Int Urogynecol J        ISSN: 0937-3462            Impact factor:   2.894


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