Literature DB >> 21443573

Neurochemical deficits in the cerebellar vermis in child offspring of parents with bipolar disorder.

Manpreet K Singh1, Daniel Spielman, Allison Libby, Elizabeth Adams, Tenah Acquaye, Meghan Howe, Ryan Kelley, Allan Reiss, Kiki D Chang.   

Abstract

OBJECTIVES: We aimed to compare concentrations of N-acetyl aspartate, myo-inositol, and other neurometabolites in the cerebellar vermis of offspring at risk for bipolar disorder (BD) and healthy controls to examine whether changes in these neuronal metabolite concentrations occur in at-risk offspring prior to the onset of mania.
METHODS: A total of 22 children and adolescents aged 9-17 years with a familial risk for bipolar I or II disorder [at-risk offspring with non-bipolar I disorder mood symptoms (AR)], and 25 healthy controls (HC) were examined using proton magnetic resonance spectroscopy at 3T to study metabolite concentrations in an 8-cc voxel in the cerebellar vermis.
RESULTS: Decreased myo-inositol and choline concentrations in the vermis were seen in the AR group compared to HC (p<0.01).
CONCLUSIONS: Decreased cellular metabolism and interference with second messenger pathways may be present in the cerebellar vermis in youth at risk for BD as evident by decreased myo-inositol and choline concentrations in this region. These results may be limited by a cross-sectional design, co-occurring diagnoses, and medication exposure. Longitudinal studies are necessary to determine whether early neurochemical changes can predict the development of mania. Improved methods for identifying children with certain neurochemical vulnerabilities may inform preventive and early intervention strategies prior to the onset of mania.
© 2011 John Wiley and Sons A/S.

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Year:  2011        PMID: 21443573      PMCID: PMC3066452          DOI: 10.1111/j.1399-5618.2011.00902.x

Source DB:  PubMed          Journal:  Bipolar Disord        ISSN: 1398-5647            Impact factor:   6.744


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