Literature DB >> 21432838

[No association between catechol-O-methyltransferase val158met polymorphism and alexithymia].

Sandra Hermes1, Jürgen Hennig, Markus Stingl, Falk Leichsenring, Frank Leweke.   

Abstract

OBJECTIVES: Reduced concentrations of dopamine in prefrontal brain structures may play a role in alexithymia. Dopamine degradation in the orbitofrontal cortex is regulated by catechol-O-methyltransferase (COMT), and a functional single nucleotide polymorphism of the COMT gene, Val158Met, has been related to psychiatric illness. This study examines the association between the COMT Val158Met gene polymorphism, and alexithymia.
METHODS: 120 healthy students and 120 patients with mental disorders were genotyped for the COMT Val158Met polymorphism. Additionally, the Toronto Alexithymia Scale (TAS-20) was administered.
RESULTS: COMT genotype did not show a significant correlation with the TAS-20 in either group.
CONCLUSIONS: COMT Val158Met polymorphism alone does not seem to be a major factor in alexithymia in healthy students. This is true even if patients with mental disorders covering a broader range of alexithymia are included. Thus, other genes, possibly interacting with cultural, environmental, and developmental factors, may be implicated.

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Year:  2011        PMID: 21432838     DOI: 10.13109/zptm.2011.57.1.51

Source DB:  PubMed          Journal:  Z Psychosom Med Psychother        ISSN: 1438-3608            Impact factor:   0.791


  1 in total

1.  Association between the Catechol-O-Methyltransferase (COMT) Val¹⁵⁸Met Polymorphism and Alexithymia in Patients with Obsessive-Compulsive Disorder.

Authors:  Min Jung Koh; Jee In Kang; Kee Namkoong; Su Young Lee; Se Joo Kim
Journal:  Yonsei Med J       Date:  2016-05       Impact factor: 2.759

  1 in total

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