| Literature DB >> 21412049 |
Carole Muller1, Robert Salvayre, Anne Nègre-Salvayre, Cécile Vindis.
Abstract
Oxidized LDLs (oxLDLs) induce various cellular dysfunctions potentially implicated in the pathogenesis of atherosclerosis. For instance, toxic concentrations of oxLDLs trigger ER stress, autophagy and apoptosis. High-density lipoproteins (HDLs) counteract several adverse biological effects triggered by oxLDLs. Our recent study reveals that HDLs inhibit the activation of ER stress and of autophagy induced by oxLDLs.Entities:
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Year: 2011 PMID： 21412049 DOI： 10.4161/auto.7.5.15003
Source DB: PubMed Journal: Autophagy ISSN： 1554-8627 Impact factor: 16.016