| Literature DB >> 2138990 |
D Mengin-Lecreulx1, J van Heijenoort.
Abstract
It was speculated that the increase of the UDP-GlcNAc pool observed with chloramphenicol can modulate the residual PEP:UDP-GlcNAc-enolpyruvate activity of fosfomycin-treated cells. This provided an explanation on how chloramphenicol can insure the formation of enough UDP-MurNAc-pentapeptide to sustain peptidoglycan synthesis at a rate that will antagonize fosfomycin-induced lysis.Entities:
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Year: 1990 PMID: 2138990 DOI: 10.1016/0378-1097(90)90270-z
Source DB: PubMed Journal: FEMS Microbiol Lett ISSN: 0378-1097 Impact factor: 2.742