Literature DB >> 21385886

Phase I and pharmacodynamic study of an orally administered novel inhibitor of histone deacetylases, SB939, in patients with refractory solid malignancies.

W P Yong1, B C Goh, R A Soo, H C Toh, K Ethirajulu, J Wood, V Novotny-Diermayr, S C Lee, W L Yeo, D Chan, D Lim, E Seah, R Lim, J Zhu.   

Abstract

BACKGROUND: The objective of this study was to assess the safety, maximum tolerated dose (MTD), pharmacokinetics, pharmacodynamics, and preliminary efficacy of SB939, a novel histone deacetylase (HDAC) inhibitor, in patients with advanced solid malignancies. PATIENTS AND METHODS: Dose-escalating cohorts of three to six patients received SB939 orally thrice weekly for 3 weeks in a 4-week cycle. Acetylated histone H3 (acH3) was measured in peripheral blood mononuclear cells (PBMCs).
RESULTS: Thirty patients treated at one of five doses (10-80 mg/day) received 79 cycles of SB939 (range, 1-12 cycles). Dose-limiting toxic effects were fatigue, hypokalemia, troponin T elevation, and QTc prolongation. Peak plasma concentration (C(max)) and area under the concentration-time curve extrapolated to infinity increased dose proportionally. The MTD of SB939 was 80 mg/day. The mean elimination half-life and oral clearance of SB939 were 7.2 ± 0.6 h and 53.0 ± 8.5 l/h, respectively, with no substantial accumulation on day 15. An increase in acH3 was observed at hour 3 and correlated with dose and C(max). Stable disease was seen in several tumor types treated at ≥40 mg. HDAC inhibition was consistently observed at 60 mg, the recommended dose.
CONCLUSIONS: SB939 can be safely administered at the recommended dose and reaches plasma levels that strongly inhibit HDAC in PBMCs. These data support further efficacy studies of SB939.

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Year:  2011        PMID: 21385886     DOI: 10.1093/annonc/mdq784

Source DB:  PubMed          Journal:  Ann Oncol        ISSN: 0923-7534            Impact factor:   32.976


  10 in total

1.  The prostate cancer blocking potential of the histone deacetylase inhibitor LBH589 is not enhanced by the multi receptor tyrosine kinase inhibitor TKI258.

Authors:  Stefan Vallo; Jens Mani; Matthias Stastny; Jasmina Makarević; Eva Juengel; Igor Tsaur; Georg Bartsch; Axel Haferkamp; Roman A Blaheta
Journal:  Invest New Drugs       Date:  2012-07-17       Impact factor: 3.850

2.  Phase 1 dose escalation multicenter trial of pracinostat alone and in combination with azacitidine in patients with advanced hematologic malignancies.

Authors:  Yasmin M Abaza; Tapan M Kadia; Elias J Jabbour; Marina Y Konopleva; Gautam Borthakur; Alessandra Ferrajoli; Zeev Estrov; William G Wierda; Ana Alfonso; Toh Han Chong; Charles Chuah; Liang-Piu Koh; Boon-Cher Goh; Julie E Chang; Daniel E Durkes; Maria Cielo Foudray; Hagop M Kantarjian; Xiao Qin Dong; Guillermo Garcia-Manero
Journal:  Cancer       Date:  2017-08-25       Impact factor: 6.860

Review 3.  New and emerging HDAC inhibitors for cancer treatment.

Authors:  Alison C West; Ricky W Johnstone
Journal:  J Clin Invest       Date:  2014-01-02       Impact factor: 14.808

4.  Antimalarial activity of the anticancer histone deacetylase inhibitor SB939.

Authors:  Subathdrage D M Sumanadasa; Christopher D Goodman; Andrew J Lucke; Tina Skinner-Adams; Ishani Sahama; Ashraful Haque; Tram Anh Do; Geoffrey I McFadden; David P Fairlie; Katherine T Andrews
Journal:  Antimicrob Agents Chemother       Date:  2012-04-16       Impact factor: 5.191

5.  A phase II study of the HDAC inhibitor SB939 in patients with castration resistant prostate cancer: NCIC clinical trials group study IND195.

Authors:  B J Eigl; S North; E Winquist; D Finch; L Wood; S S Sridhar; J Powers; J Good; M Sharma; J A Squire; J Bazov; T Jamaspishvili; M E Cox; P A Bradbury; E A Eisenhauer; K N Chi
Journal:  Invest New Drugs       Date:  2015-05-19       Impact factor: 3.850

6.  Phase I study of CKD-581, a pan-histone deacetylase inhibitor, in patients with lymphoma or multiple myeloma refractory to standard therapy.

Authors:  Hyungwoo Cho; Dok Hyun Yoon; Kyu-Pyo Kim; Kyun-Seop Bae; Won Seog Kim; Hyeon-Seok Eom; Jin Seok Kim; Jung Yong Hong; Seok Jin Kim; Hyewon Lee; Soo-Jeong Kim; Cheolwon Suh
Journal:  Invest New Drugs       Date:  2018-03-09       Impact factor: 3.651

Review 7.  Epigenetic treatment of solid tumours: a review of clinical trials.

Authors:  Clara Nervi; Elisabetta De Marinis; Giovanni Codacci-Pisanelli
Journal:  Clin Epigenetics       Date:  2015-12-10       Impact factor: 6.551

8.  Role of Hydroxamate-Based Histone Deacetylase Inhibitors (Hb-HDACIs) in the Treatment of Solid Malignancies.

Authors:  Antonino Grassadonia; Pasquale Cioffi; Felice Simiele; Laura Iezzi; Marinella Zilli; Clara Natoli
Journal:  Cancers (Basel)       Date:  2013-07-25       Impact factor: 6.639

9.  Identification of brain metastasis genes and therapeutic evaluation of histone deacetylase inhibitors in a clinically relevant model of breast cancer brain metastasis.

Authors:  Soo-Hyun Kim; Richard P Redvers; Lap Hing Chi; Xiawei Ling; Andrew J Lucke; Robert C Reid; David P Fairlie; Ana Carolina Baptista Moreno Martin; Robin L Anderson; Delphine Denoyer; Normand Pouliot
Journal:  Dis Model Mech       Date:  2018-07-06       Impact factor: 5.758

10.  Activity of histone deacetylase inhibitors and an Aurora kinase inhibitor in BCR-ABL-expressing leukemia cells: Combination of HDAC and Aurora inhibitors in BCR-ABL-expressing cells.

Authors:  Seiichi Okabe; Tetsuzo Tauchi; Yuko Tanaka; Shinya Kimura; Taira Maekawa; Kazuma Ohyashiki
Journal:  Cancer Cell Int       Date:  2013-04-04       Impact factor: 5.722

  10 in total

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