Literature DB >> 21360513

Efficacy and safety of anakinra therapy in pediatric and adult patients with the autoinflammatory Muckle-Wells syndrome.

Jasmin B Kuemmerle-Deschner1, Pascal N Tyrrell, Ina Koetter, Helmut Wittkowski, Anja Bialkowski, Nicolai Tzaribachev, Peter Lohse, Assen Koitchev, Christoph Deuter, Dirk Foell, Susanne M Benseler.   

Abstract

OBJECTIVE: Muckle-Wells syndrome (MWS) is an inherited autoinflammatory disease caused by mutations in the NLRP3 gene that result in excessive interleukin-1 (IL-1) release. It is characterized by severe fevers, rashes, arthralgia, and conjunctivitis, leading to sensorineural deafness and amyloidosis. The recombinant IL-1 receptor antagonist anakinra blocks the biologic activity of IL-1. The aim of this study was to determine the short- and long-term efficacy and safety of anakinra therapy in children and adults with severe MWS.
METHODS: A single-center observational study was performed. Standardized assessments included clinical features, the Disease Activity Score (DAS) for MWS, classic and novel markers of inflammation, and patient-derived measures of health status. The primary outcome was a score of <10 on the DAS for MWS at 2 weeks and at the last followup visit. Measures of MWS disease activity were investigated using descriptive statistics and paired comparative analysis.
RESULTS: A total of 12 patients with severe MWS (5 children and 7 adults) received anakinra for a median of 11 months (range 5-14 months). The median followup was 11 months (range 5-14 months). Disease activity was significantly lower in all patients at 2 weeks (P = 0.0005). Organ manifestations of MWS improved, as did all patient-derived measures of health status, markers of inflammation, and hearing loss in 2 of the patients. Levels of the novel neutrophil activation biomarker S100A12 followed clinical disease activity. Treatment was well tolerated, and no serious adverse events were observed.
CONCLUSION: Anakinra was found to be a safe and effective treatment of severe MWS, leading to a significant improvement in disease activity at 2 weeks as well as long-term. Anakinra therapy should therefore be considered in children and adults with severe MWS disease requiring IL-1 blockade.
Copyright © 2011 by the American College of Rheumatology.

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Year:  2011        PMID: 21360513     DOI: 10.1002/art.30149

Source DB:  PubMed          Journal:  Arthritis Rheum        ISSN: 0004-3591


  56 in total

Review 1.  Type I IFN-mediated regulation of IL-1 production in inflammatory disorders.

Authors:  Kristina Ludigs; Valeriy Parfenov; Renaud A Du Pasquier; Greta Guarda
Journal:  Cell Mol Life Sci       Date:  2012-04-24       Impact factor: 9.261

Review 2.  Immunology in clinic review series; focus on autoinflammatory diseases: update on monogenic autoinflammatory diseases: the role of interleukin (IL)-1 and an emerging role for cytokines beyond IL-1.

Authors:  R Goldbach-Mansky
Journal:  Clin Exp Immunol       Date:  2012-03       Impact factor: 4.330

Review 3.  The emerging role of interleukin-1β in autoinflammatory diseases.

Authors:  Thirusha Lane; Helen J Lachmann
Journal:  Curr Allergy Asthma Rep       Date:  2011-10       Impact factor: 4.806

Review 4.  Anakinra as an interleukin 1 receptor antagonist, complicated genetics and molecular impacts--from the point of view of mouse genomics.

Authors:  Yanhong Cao; Yan Jiao; Lishi Wang; Yue Huang; Arnold Postlethwaite; John Stuart; Andy Kang; Robert W Williams; Weikuan Gu
Journal:  Int Immunopharmacol       Date:  2012-03-14       Impact factor: 4.932

5.  A case of cryopyrin-associated periodic syndrome with kidney transplant failure.

Authors:  Rie Imaizumi; Yasuo Ishii; Katsuyuki Miki; Shinji Kudo; Toru Murakami; Ichiro Koyama; Ichiro Nakajima; Shohei Fuchinoue; Yasushi Kawaguchi; Atsuo Taniguchi; Satoshi Teraoka
Journal:  CEN Case Rep       Date:  2014-06-14

Review 6.  [Autoinflammatory syndromes : Practical approach to diagnostics and therapy].

Authors:  J B Kümmerle-Deschner
Journal:  Z Rheumatol       Date:  2016-08       Impact factor: 1.372

7.  NLRP3 mutation and cochlear autoinflammation cause syndromic and nonsyndromic hearing loss DFNA34 responsive to anakinra therapy.

Authors:  Hiroshi Nakanishi; Yoshiyuki Kawashima; Kiyoto Kurima; Jae Jin Chae; Astin M Ross; Gineth Pinto-Patarroyo; Seema K Patel; Julie A Muskett; Jessica S Ratay; Parna Chattaraj; Yong Hwan Park; Sriharsha Grevich; Carmen C Brewer; Michael Hoa; H Jeffrey Kim; John A Butman; Lori Broderick; Hal M Hoffman; Ivona Aksentijevich; Daniel L Kastner; Raphaela Goldbach-Mansky; Andrew J Griffith
Journal:  Proc Natl Acad Sci U S A       Date:  2017-08-28       Impact factor: 11.205

Review 8.  Review: Found in Translation: International Initiatives Pursuing Interleukin-1 Blockade for Treatment of Acute Kawasaki Disease.

Authors:  Jane C Burns; Isabelle Koné-Paut; Taco Kuijpers; Chisato Shimizu; Adriana Tremoulet; Moshe Arditi
Journal:  Arthritis Rheumatol       Date:  2017-02       Impact factor: 10.995

9.  Rationale and study design for a phase I/IIa trial of anakinra in children with Kawasaki disease and early coronary artery abnormalities (the ANAKID trial).

Authors:  Adriana H Tremoulet; Sonia Jain; Susan Kim; Jane Newburger; Moshe Arditi; Alessandra Franco; Brookie Best; Jane C Burns
Journal:  Contemp Clin Trials       Date:  2016-04-11       Impact factor: 2.226

10.  Localization and functionality of the inflammasome in neutrophils.

Authors:  Martina Bakele; Melanie Joos; Sofia Burdi; Nicolas Allgaier; Simone Pöschel; Birgit Fehrenbacher; Martin Schaller; Veronica Marcos; Jasmin Kümmerle-Deschner; Nikolaus Rieber; Niels Borregaard; Amir Yazdi; Andreas Hector; Dominik Hartl
Journal:  J Biol Chem       Date:  2014-01-07       Impact factor: 5.157

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