Literature DB >> 21346218

Defining hematoma expansion in intracerebral hemorrhage: relationship with patient outcomes.

D Dowlatshahi1, A M Demchuk, M L Flaherty, M Ali, P L Lyden, E E Smith.   

Abstract

BACKGROUND: Hematoma expansion (HE) is a surrogate marker in intracerebral hemorrhage (ICH) trials. However, the amount of HE necessary to produce poor outcomes in an individual is unclear; there is no agreement on a clinically meaningful definition of HE. We compared commonly used definitions of HE in their ability to predict poor outcome as defined by various cutpoints on the modified Rankin Scale (mRS).
METHODS: In this cohort study, we analyzed 531 patients with ICH from the Virtual International Stroke Trials Archive. Primary outcome was mRS at 90 days, dichotomized into 0-3 vs 4-6. Secondary outcomes included other mRS cutpoints and mRS "shift analysis." Sensitivity, specificity, and predictive values for commonly used HE definitions were calculated.
RESULTS: Between 13% and 32% of patients met the commonly used HE definitions. All definitions independently predicted poor outcome; positive predictive values increased with higher growth cutoffs but at the expense of lower sensitivities. All HE definitions showed higher specificity than sensitivity. Absolute growth cutoffs were more predictive than relative cutoffs when mRS 5-6 or 6 was defined as "poor outcome."
CONCLUSION: HE robustly predicts poor outcome regardless of the growth definition or the outcome definition. The highest positive predictive values are obtained when using an absolute growth definition to predict more severe outcomes. Given that only a minority of patients may have clinically relevant HE, hemostatic ICH trials may need to enroll a large number of patients, or select for a population that is more likely to have HE.

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Year:  2011        PMID: 21346218      PMCID: PMC3068004          DOI: 10.1212/WNL.0b013e3182143317

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


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2.  Predisposing factors to enlargement of spontaneous intracerebral hematoma.

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4.  Multivariate analysis of predictors of hematoma enlargement in spontaneous intracerebral hemorrhage.

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5.  Enlargement of spontaneous intracerebral hemorrhage. Incidence and time course.

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6.  Efficacy and safety of recombinant activated factor VII for acute intracerebral hemorrhage.

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7.  Defining the CT angiography 'spot sign' in primary intracerebral hemorrhage.

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10.  Early care limitations independently predict mortality after intracerebral hemorrhage.

Authors:  D B Zahuranec; D L Brown; L D Lisabeth; N R Gonzales; P J Longwell; M A Smith; N M Garcia; L B Morgenstern
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2.  Apolipoprotein E genotype predicts hematoma expansion in lobar intracerebral hemorrhage.

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3.  Refining Prognosis for Intracerebral Hemorrhage by Early Reassessment.

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4.  Leukocyte Count and Intracerebral Hemorrhage Expansion.

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5.  Clotting factors to treat thrombolysis-related symptomatic intracranial hemorrhage in acute ischemic stroke.

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Review 6.  Update on the Treatment of Spontaneous Intraparenchymal Hemorrhage: Medical and Interventional Management.

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7.  Clinical Outcomes and Neuroimaging Profiles in Nondisabled Patients With Anticoagulant-Related Intracerebral Hemorrhage.

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8.  Integration of Computed Tomographic Angiography Spot Sign and Noncontrast Computed Tomographic Hypodensities to Predict Hematoma Expansion.

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9.  Lactate Dehydrogenase Predicts Early Hematoma Expansion and Poor Outcomes in Intracerebral Hemorrhage Patients.

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Review 10.  What does the CT angiography "spot sign" of intracerebral hemorrhage mean in modern neurosurgical settings with minimally invasive endoscopic techniques?

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