Literature DB >> 21273600

Effect of genistein on p90RSK phosphorylation and cell proliferation in T47D breast cancer cells.

John Gwin1, Neil Drews, Syed Ali, Justin Stamschror, Matthew Sorenson, Talitha T Rajah.   

Abstract

BACKGROUND: The molecular mechanisms of genistein's proliferative effects on breast cancer cells are largely unknown. This study aimed to examine estrogen-receptor (ER)-related signaling molecules involved in genistein-associated cell proliferation and survival (ERK1/2, p90RSK, JNK, Akt and NFκB) and to correlate these results to cell proliferation.
MATERIALS AND METHODS: The effect of genistein on cell-signaling molecules was determined in T47D breast cancer cells by a Bioplex phosphoprotein detection kit. These results were confirmed by Western blotting and were correlated to cell proliferation by MTT assay.
RESULTS: Low and high concentrations of genistein induced an ERK1/2-independent decrease in phosphorylated p90RSK. This effect was accompanied by decreased cell proliferation at high concentrations and an increased response at low concentrations of genistein following a 48-hour exposure.
CONCLUSION: Concentration-dependent actions of genistein in T47D cells may be due to differential activation of signaling molecules.

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Year:  2011        PMID: 21273600

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  6 in total

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3.  Inhibitory Effect of Genistein on PLC/PRF5 Hepatocellular Carcinoma Cell Line.

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Journal:  Cancers (Basel)       Date:  2018-09-21       Impact factor: 6.639

6.  BRAF inhibition curtails IFN-gamma-inducible PD-L1 expression and upregulates the immunoregulatory protein galectin-1 in melanoma cells.

Authors:  Patryk Górniak; Maja Wasylecka-Juszczyńska; Iwona Ługowska; Piotr Rutkowski; Anna Polak; Maciej Szydłowski; Przemysław Juszczyński
Journal:  Mol Oncol       Date:  2020-05-19       Impact factor: 6.603

  6 in total

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